感觉神经肽与气道内脂质介质的相互作用。

Journal of lipid mediators Pub Date : 1993-10-01
S Manzini, F Perretti, S Meini
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引用次数: 0

摘要

在哺乳动物气道中,辣椒素敏感的初级传入神经中感觉神经肽的局部释放引起了突出的运动和炎症反应。这种神经源性炎症可导致哮喘和气道高反应性的病理生理。在这篇综述中,证据将提出关于这条肽能神经通路在脂质介质(如类二十烷酸和PAF)的一些肺作用中的介导作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Interactions between sensory neuropeptides and lipid mediators in the airways.

The local release of sensory neuropeptides from capsaicin-sensitive primary afferents elicits prominent motor and inflammatory actions in mammalian airways. This neurogenic inflammation can contribute to the pathophysiology of asthma and airway hyperreactivity. In this review evidence will be presented regarding the involvement of this peptidergic neural pathway in the mediation of some pulmonary actions of lipid mediators such as eicosanoids and PAF.

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