[潜水者短期下潜至100米后外呼吸静、动态特征的变化]。

Fiziologicheskiĭ zhurnal Pub Date : 1993-09-01
V M Ilin, S O Huliar, A I Dmitruk
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引用次数: 0

摘要

在巴伦采夫海的条件下,在深度100米的深度下进行了一系列短潜(不考虑减压时间,最多45分钟)(SD),对5名专业深海潜水员进行了测试。在深潜至40米时,使用压缩空气作为呼吸介质;在深潜至60、80和100米时,使用氮气-氦气混合物。在SD系列之前,在单独的SD之间以及潜水至100 m减压结束后的第3天,使用计算机肺活量和气测法测量静息时和在空气和螺旋介质中强制呼气时的肺体积和速度变量。支气管痉挛综合征在短暂性孤立性或全身性气道阻塞的发展中起主导作用,在SD系列传导过程中被证明在潜水员中被诱发。推测梗阻现象与肺组织弹性特性的紊乱和呼气萎陷的发生有关;它们可能引起持续的肺扩张。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Changes of static and dynamic characteristics of external respiration in divers after short-term dives down to 100 m].

In a series of short (up to 45 min without considering time of decompression) dives (SD) at depths down to 100 m under conditions of the Barentsev Sea five professional deep-sea divers were examined. In dives down to 40 m compressed air was used as breathing media, in dives to 60, 80 and 100 m--nitrogen-heliox mixture. Before the SD series the volume and velocity lung variables at rest and during forced expiration in air and heliox media were measured between separate SDs and on the 3rd day after the end of decompression with dive to 100 m, using computer spiro- and pneumotachometry methods. Bronchospastic syndrome, playing the leading role in the development of transient isolated or generalized airway obstruction, was shown to be evoked in divers during conduction of SD series. Obstructive phenomena are presumed to be connected with elastic properties' disturbance of lung tissue and development of expiratory collapse; they might cause continuous lung dilatation.

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