[正常状态和心功能不全各阶段人心肌收缩力和能量代谢]。

Fiziologicheskiĭ zhurnal Pub Date : 1993-09-01
V H Shevchuk, S B Frantsuzova, N O Horchakova, O I Plyska, V V Babak, L L Arshynnykova, V V Lazoryshynets, V V Iakymovych
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引用次数: 0

摘要

在对照组和心力衰竭发展的早期阶段,研究了心肌的收缩功能和代谢。心力衰竭早期心肌收缩功能的支持是通过大量使用大能磷酸盐和它们的不充分再合成来提供的。在心肌衰竭晚期,由于高能磷酸盐转运到其利用部位受到干扰,心肌松弛恶化。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Contractility and energy metabolism in human myocardium in normal state and in various stages of cardiac insufficiency].

Contractile function and metabolism of byoptates of human myocardium were investigated in the control group and at early stages of heart failure development. Support of the myocardial contractile function at the early stage of the heart failure is provided by intensive use of macroergic phosphates and their insufficient resynthesis. At the late stages of myocardial failure deterioration of the heart muscle relaxation was observed as a result of disturbances in transport of high-ergic phosphates to the sites of their utilization.

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