甲基营养细菌的溶原性和溶原性转化。2兼性甲醇同化醋酸杆菌菌株的溶原转化[j]。

L Wünsche, B Kiesel, H Fischer
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引用次数: 4

摘要

甲基营养菌株醋酸杆菌MB 58/1的溶菌状态通过固化和溶菌实验得到了完整的证实。在这些研究中,我们发现一些表型特征被存在或缺失的噬菌体mo1所改变。结果表明,噬菌体的血清学行为、对噬菌体的吸附和对土霉素的敏感性的变化是由溶原转化引起的。噬菌体基因组诱导的细菌细胞表型改变是细胞表面脂多糖结构修饰的结果。在土霉素耐药的情况下,必须假设修饰的脂多糖结构与细胞膜的特定运输蛋白之间的相互作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Lysogeny and lysogenic conversion in methylotrophic bacteria. II. Lysogenic conversion in facultative methanol-assimilating Acetobacter strains].

The lysogenic state of the methylotrophic strain Acetobacter MB 58/1 is completely demonstrated by curing and lysogenization experiments. During these investigations we found that some phenotypic characteristics are modified by the presence or loss of the prophage MO 1. It could be shown that changes of the serological behaviour, the adsorption of the phages and the sensitivity against oxytetracycline are caused by lysogenic conversion. The phenotypic alterations of the bacterial cells induced by the phage genome are the result of modifications of the lipopolysaccharide structures on the cell surface. In the case of oxytetracycline resistance, interactions between the modified lipopolysaccharide structures and specific transport proteins of the cell membranes must be assumed.

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