{"title":"脑卒中与胶质母细胞瘤相关。","authors":"B H Dobkin","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>Several months after onset of typical stroke syndromes, two patients were found to harbor a glioblastoma multiforme in the area of infarction. Arteriographic and late CT studies suggested that one neoplasm caused parenchymal compressive occlusion of a sylvian branch mimicking embolism and the other entwined the origin of the middle cerebral artery in the leptomeninges simulating atherothrombosis.</p>","PeriodicalId":77682,"journal":{"name":"Bulletin of clinical neurosciences","volume":null,"pages":null},"PeriodicalIF":0.0000,"publicationDate":"1985-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Stroke associated with glioblastoma.\",\"authors\":\"B H Dobkin\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>Several months after onset of typical stroke syndromes, two patients were found to harbor a glioblastoma multiforme in the area of infarction. Arteriographic and late CT studies suggested that one neoplasm caused parenchymal compressive occlusion of a sylvian branch mimicking embolism and the other entwined the origin of the middle cerebral artery in the leptomeninges simulating atherothrombosis.</p>\",\"PeriodicalId\":77682,\"journal\":{\"name\":\"Bulletin of clinical neurosciences\",\"volume\":null,\"pages\":null},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1985-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Bulletin of clinical neurosciences\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Bulletin of clinical neurosciences","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Several months after onset of typical stroke syndromes, two patients were found to harbor a glioblastoma multiforme in the area of infarction. Arteriographic and late CT studies suggested that one neoplasm caused parenchymal compressive occlusion of a sylvian branch mimicking embolism and the other entwined the origin of the middle cerebral artery in the leptomeninges simulating atherothrombosis.
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