超越淀粉样蛋白:纳米体介导的阿尔茨海默病神经炎症治疗。

IF 15.2 1区 医学 Q1 NEUROSCIENCES
Soukaina Amniouel, Jessica Suh, Wei Zheng, Qi Zhang
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引用次数: 0

摘要

阿尔茨海默病(AD)是最常见和最具破坏性的神经退行性疾病之一,其特征是淀粉样β (Aβ)斑块的积累、tau蛋白的神经原纤维缠结和神经炎症的持续存在,导致进行性认知能力下降、独立性丧失、家庭情绪和经济压力以及重大的社会成本。目前的抗淀粉样蛋白治疗在去除β淀粉样蛋白方面部分成功,但往往会导致炎症增加。这导致治疗效果有限,并引起淀粉样蛋白相关成像异常等副作用。此外,它们不能解决阿尔茨海默病患者的神经炎症。在这篇综述中,我们讨论了一种新的治疗策略,即结合抗a β原纤维的单域抗体(sabs,纳米体)和抗炎药物,并将它们应用于与AD患者斑块相关的神经炎症区域。该策略旨在控制活化的小胶质细胞和星形胶质细胞的功能,从而避免不必要的免疫抑制。我们还讨论了sabs的独特特征,包括小尺寸,良好的组织穿透性,缺乏fc介导的免疫反应,以及相关的有效载荷(即小分子,生物制剂和纳米颗粒)和递送系统。这种免疫调节疗法专门针对斑块,因此代表了一个有希望的机会,以提高淀粉样蛋白清除和针对AD的炎症成分,潜在地提高疾病的治疗效果。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Beyond amyloid: nanobody-mediated neuroinflammatory therapy for Alzheimer's disease.

Alzheimer's disease (AD) is one of the most common and devastating neurodegenerative diseases, characterized by accumulation of amyloid-beta (Aβ) plaques, neurofibrillary tangles of tau protein, and persistence of neuroinflammation, leading to progressive cognitive decline, loss of independence, emotional and financial strain on families, and significant societal costs. Current anti-amyloid treatments are partly successful in removing Aβ amyloid, but often lead to increased inflammation. This leads to limited therapeutic efficacy and causes side effects such as amyloid-related imaging abnormalities. In addition, they do not address neuroinflammation in AD patients. In this review, we discuss a new therapeutic strategy that combines single-domain antibodies (sdAbs, nanobodies) against Aβ fibrils and anti-inflammatory drugs and applies them to the regions of neuroinflammation associated with the plaques in AD patients. This strategy aims to control the function of activated microglia and astrocytes, thereby avoiding unnecessary immunosuppression. We also discuss the unique features of sdAbs, including small size, good tissue penetration, and lack of Fc-mediated immune reactions, as well as relevant payloads (i.e., small molecules, biologics, and nanoparticles) and delivery systems. This immunomodulatory therapy targets the plaques specifically and therefore represents a promising opportunity to improve amyloid clearance and target the inflammatory components of AD, potentially improving the therapeutic efficacy of the disease.

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来源期刊
Translational Neurodegeneration
Translational Neurodegeneration Neuroscience-Cognitive Neuroscience
CiteScore
19.50
自引率
0.80%
发文量
44
审稿时长
10 weeks
期刊介绍: Translational Neurodegeneration, an open-access, peer-reviewed journal, addresses all aspects of neurodegenerative diseases. It serves as a prominent platform for research, therapeutics, and education, fostering discussions and insights across basic, translational, and clinical research domains. Covering Parkinson's disease, Alzheimer's disease, and other neurodegenerative conditions, it welcomes contributions on epidemiology, pathogenesis, diagnosis, prevention, drug development, rehabilitation, and drug delivery. Scientists, clinicians, and physician-scientists are encouraged to share their work in this specialized journal tailored to their fields.
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