失血性休克时糖萼损伤需要铁下垂途径。

IF 2.9 3区医学 Q2 CRITICAL CARE MEDICINE

SHOCK Pub Date : 2025-09-05 DOI:10.1097/SHK.0000000000002701

Aditya Vinjamuri, David Engelhardt, Willard R Covey, Sarah Abdullah, Farhana Shaheen, Zander Gerberg, Sharven Taghavi, Juan Duchesne, Olan Jackson-Weaver

引用次数: 0

摘要

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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The ferroptosis pathway is required for glycocalyx damage during hemorrhagic shock.

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来源期刊

SHOCK 医学-外科

CiteScore

6.20

自引率

3.20%

发文量

199

审稿时长

1 months

期刊介绍： SHOCK®: Injury, Inflammation, and Sepsis: Laboratory and Clinical Approaches includes studies of novel therapeutic approaches, such as immunomodulation, gene therapy, nutrition, and others. The mission of the Journal is to foster and promote multidisciplinary studies, both experimental and clinical in nature, that critically examine the etiology, mechanisms and novel therapeutics of shock-related pathophysiological conditions. Its purpose is to excel as a vehicle for timely publication in the areas of basic and clinical studies of shock, trauma, sepsis, inflammation, ischemia, and related pathobiological states, with particular emphasis on the biologic mechanisms that determine the response to such injury. Making such information available will ultimately facilitate improved care of the traumatized or septic individual.