氨基甲酰化促进α-Synuclein KTKEGV重复基序的序列特异性淀粉样蛋白形成。

IF 3.9 3区 医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY
Joshna Gadhavi, , , Sumedha Shah, , , Mohini Patel, , and , Sharad Gupta*, 
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引用次数: 0

摘要

KTKEGV重复基序在调节α-突触核蛋白折叠的结构和功能,包括四聚体的形成和与膜的相互作用中起着至关重要的作用。在α-syn中,已经报道了9个KTKEGV不完全重复序列。翻译后修饰(PTMs)可以中和赖氨酸上的正电荷,干扰α-syn与细胞膜的静电相互作用,从而影响生理功能。氨甲酰化是一种非酶促年龄依赖性电荷中和PTM,据报道在老年人中升高。虽然一些报道认为氨甲酰化是一种促聚集因子,但也有人认为它具有保护作用。在本研究中,我们探索了KTKEGV重复基序和全长α-syn蛋白在氨甲酰化后的聚集倾向。我们观察到α-syn的各种KTKEGV基元之间不同的聚集动力学,包括疾病特异性突变版本,这通过多种生物物理技术(如ThT测定、浊度测量、刚果红染色、AFM和SEM)得到证实。值得注意的是,重复基序3 (32KTKEGVLYV40)、5 (58KTKEQVTNV66)和核心基序(77VAQKTV82)在氨基酰化时表现出强大的纤维淀粉样蛋白形成。此外,重复基序4 (43KTKEGVVH50)在发生突变(E46K, H50Q,以及两者)时,成为一个聚集热点,即使天然序列在氨甲酰化时不聚集。ThT平台荧光增强数倍表明,全长α-syn蛋白的氨甲酰化似乎导致纤维淀粉样蛋白含量较高的聚集。这种氨甲酰化的α-syn聚集体可以招募未修饰的α-syn,并再次导致组织良好的淀粉样原纤维的形成。这些发现突出了电荷中和型ptm的位点特异性作用,如氨甲酰化α-syn聚集,并为突触核蛋白病和路易小体痴呆相关的分子机制提供了新的见解。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Carbamylation Promotes Sequence-Specific Amyloidogenesis in the α-Synuclein KTKEGV Repeat Motifs

Carbamylation Promotes Sequence-Specific Amyloidogenesis in the α-Synuclein KTKEGV Repeat Motifs

KTKEGV repeat motifs play a crucial role in regulating the folding of α-synuclein structure and function, including tetramer formation and interaction with membranes. In α-syn, nine KTKEGV imperfect repeats have been reported. Post-translational modifications (PTMs) that could neutralize the positive charge on lysine may disturb the electrostatic interaction of α-syn with the cellular membrane, thereby affecting physiological function. Carbamylation is one such nonenzymatic age-dependent charge-neutralizing PTM which is reported to be elevated in aged people. While some reports have suggested that carbamylation is a proaggregation factor, others have assigned it a protective role. In the present study, we explored the aggregation propensities of KTKEGV repeat motifs and full-length α-syn protein upon carbamylation. We observed distinct aggregation kinetics among various KTKEGV motifs of α-syn, including disease-specific mutated versions, which was confirmed by multiple biophysical techniques such as ThT assay, turbidity measurement, Congo red staining, AFM, and SEM. Notably, the repeat motifs 3 (32KTKEGVLYV40), 5 (58KTKEQVTNV66), and the core motif (77VAQKTV82) exhibited robust fibrillar amyloid formation when carbamylated. Also, repeat motif 4 (43KTKEGVVH50), when mutated (E46K, H50Q, and both), becomes an aggregation hotspot, even though the native sequence does not aggregate upon carbamylation. Carbamylation of full-length α-syn protein appears to lead to aggregation with higher fibrillar amyloid content as indicated by a several-fold enhancement of ThT plateau fluorescence. Such carbamylated α-syn aggregates could recruit unmodified α-syn and again led to the formation of well-organized amyloid fibrils. These findings highlight the site-specific role of charge-neutralizing PTMs such as carbamylation α-syn aggregation and provide novel insights into the molecular mechanisms related to synucleinopathies and dementia with Lewy bodies.

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来源期刊
ACS Chemical Neuroscience
ACS Chemical Neuroscience BIOCHEMISTRY & MOLECULAR BIOLOGY-CHEMISTRY, MEDICINAL
CiteScore
9.20
自引率
4.00%
发文量
323
审稿时长
1 months
期刊介绍: ACS Chemical Neuroscience publishes high-quality research articles and reviews that showcase chemical, quantitative biological, biophysical and bioengineering approaches to the understanding of the nervous system and to the development of new treatments for neurological disorders. Research in the journal focuses on aspects of chemical neurobiology and bio-neurochemistry such as the following: Neurotransmitters and receptors Neuropharmaceuticals and therapeutics Neural development—Plasticity, and degeneration Chemical, physical, and computational methods in neuroscience Neuronal diseases—basis, detection, and treatment Mechanism of aging, learning, memory and behavior Pain and sensory processing Neurotoxins Neuroscience-inspired bioengineering Development of methods in chemical neurobiology Neuroimaging agents and technologies Animal models for central nervous system diseases Behavioral research
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