肿瘤治疗场改变胶质母细胞瘤患者局部凝血动力学。

IF 6.9 2区 医学 Q1 CLINICAL NEUROLOGY
Claudia Maletzki, Thomas Freitag, Adrian Hempelmann, Annabell Wolff, Thomas M Freiman, Sae-Yeon Won, Dirk Koczan, Sina Sender, Pablo A Valdes, Joshua D Bernstock, Florian Gessler, Daniel Dubinski
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引用次数: 0

摘要

胶质母细胞瘤(GBM)是一种高度侵袭性的脑肿瘤,与高凝性和血栓形成有关。肿瘤治疗电场(TTFields)是一种非侵入性疗法,它使用低强度的交变电场来破坏癌细胞分裂,如果与放化疗联合使用,可以延长生存期。接受TTFields治疗的患者通常表现出明显的复发模式,这表明TTFields与肿瘤相关凝血之间存在局部相互作用,其潜在机制尚不清楚。本研究通过分子、止血学和表型分析,检测了ttfields治疗患者血液、肿瘤细胞和血浆来源的细胞外囊泡的凝血情况。我们的研究结果表明,短期暴露于TTFields可通过改变组织因子(TF)表达和破坏外源性凝血途径,显著延长GBM患者和健康供者的凝血时间。TTFields通过降低因子II/FXIII活性和血小板计数来降低凝块硬度,而不损害纤维蛋白原功能。暴露于TTFields的患者源性GBM细胞表现出TF丰度增加。基于rna的微阵列分析证实了GBM细胞的凝血相关变化,包括血小板粘附标志物ITGA2上调,THBS1下调,THBS1是凝血、血小板聚集、细胞外基质重塑和肿瘤侵袭的调节因子。此外,TTFields暴露后,凝血调节基因TXNIP下调,表明与肿瘤微环境中的免疫调节有关。在患者源性GBM细胞和外周血的异体共培养模型中,TTFields调节凝血和免疫反应,可能是通过重新平衡肿瘤微环境中的促凝因子和抗凝因子,减少血栓形成前状态,改变炎症途径。这些发现为TTFields如何影响凝血并最终影响免疫调节提供了见解,为优化临床决策和减轻GBM患者的血栓栓塞并发症提供了策略。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Tumor treating fields alter local coagulation dynamics in glioblastoma patients.

Glioblastoma (GBM) is a highly aggressive brain tumor, associated with hypercoagulability and thrombosis. Tumor Treating Fields (TTFields), a non-invasive therapy that uses low-intensity, alternating electric fields to disrupt cancer cell division, prolongs survival when used concomitantly with radiochemotherapy. TTFields-treated patients often exhibit distinct recurrence patterns, suggesting a local interaction between TTFields and tumor-associated coagulation, underlying mechanisms remain unclear. This study examined coagulation profiles in TTFields-treated patients' blood, tumor cells, and plasma-derived extracellular vesicles using molecular, hemostaseologic, and phenotypic analyses. Our findings revealed that short-term TTFields exposure significantly prolongs blood coagulation in GBM patients and healthy donors by altering tissue factor (TF) expression and disrupting the extrinsic coagulation pathway. TTFields reduced clot rigidity by decreasing Factor II/FXIII activity and platelet count, without impairing fibrinogen function. Patient-derived GBM cells exposed to TTFields exhibited increased TF abundance. RNA-based microarray analysis of GBM cells confirmed coagulation-related changes, including upregulation of platelet adhesion marker ITGA2, and downregulation of THBS1, a regulator of clotting, platelet aggregation, extracellular matrix remodeling, and tumor invasiveness. Additionally, TXNIP, a coagulation-modulating gene, was downregulated after TTFields exposure, indicating a link to immune regulation in the tumor microenvironment. In an allogeneic co-culture model of patient-derived GBM cells and peripheral blood, TTFields modulated coagulation and immune responses, likely by rebalancing pro- and anticoagulant factors in the tumor microenvironment, reducing the prothrombotic state, and altering inflammatory pathways. These findings provide insights into how TTFields influence coagulation and, eventually, immune regulation, offering strategies to optimize clinical decision-making and mitigating thromboembolic complications in GBM patients.

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来源期刊
Neurotherapeutics
Neurotherapeutics 医学-神经科学
CiteScore
11.00
自引率
3.50%
发文量
154
审稿时长
6-12 weeks
期刊介绍: Neurotherapeutics® is the journal of the American Society for Experimental Neurotherapeutics (ASENT). Each issue provides critical reviews of an important topic relating to the treatment of neurological disorders written by international authorities. The Journal also publishes original research articles in translational neuroscience including descriptions of cutting edge therapies that cross disciplinary lines and represent important contributions to neurotherapeutics for medical practitioners and other researchers in the field. Neurotherapeutics ® delivers a multidisciplinary perspective on the frontiers of translational neuroscience, provides perspectives on current research and practice, and covers social and ethical as well as scientific issues.
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