外泌体小rna调控急性肺损伤/急性呼吸窘迫综合征发病机制的研究进展

Q3 Medicine
Lingshuang Chen, Jun Lyu, Xianming Zhang
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引用次数: 0

摘要

急性肺损伤/急性呼吸窘迫综合征(ALI/ARDS)是一种进展迅速、病死率高的严重危重症。它由直接/间接的肺相关或全身触发引起,导致肺上皮细胞和内皮细胞的广泛损伤。其发病机制包括由于血管渗漏和上皮损伤引起的不受控制的炎症和肺血气屏障的破坏。目前对急性呼吸窘迫综合征/急性呼吸窘迫综合征的管理仍主要是支持性的,提供症状缓解,但预后改善有限,需要更深入地探索上游致病机制,以确定更安全、更有效的治疗方法。外泌体microRNAs (miRNA)是细胞外小泡(40-150 nm),含有非编码单链rna,调节转录后细胞过程并参与ALI/ARDS病理生理。研究表明外泌体将蛋白质、核酸和mirna转运到受体细胞,介导细胞间通讯。在ALI/ARDS模型中,外泌体mirna传递给肺泡上皮细胞、内皮细胞、巨噬细胞和中性粒细胞,可严重调节自噬、焦亡、凋亡、增殖、炎症信号、巨噬细胞极化和中性粒细胞活化,从而加剧或减轻疾病进展。工程技术的最新进展通过克服天然外泌体的局限性,增强了外泌体mirna的治疗潜力。本文综述了外泌体mirna介导的ALI/ARDS发病机制在关键细胞类型中的调控,为新的治疗策略提供见解。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Research advances of exosomal micrornas in regulating the pathogenesis of acute lung injury/acute respiratory distress syndrome].

Acute lung injury/acute respiratory distress syndrome (ALI/ARDS) is a severe critical condition marked by rapid progression and high fatality. It results from direct/indirect lung-related or systemic triggers, leading to widespread injury of lung epithelial and endothelial cells. Its pathogenesis involves uncontrolled inflammation and breakdown of the lung's blood-air barrier due to leaky blood vessels and epithelial damage. Current management of ALI/ARDS remains primarily supportive, offering symptomatic relief but limited improvement in prognosis, necessitating deeper exploration of upstream pathogenic mechanisms to identify safer and more effective therapies. Exosomal microRNAs (miRNA), small extracellular vesicles (40-150 nm) containing non-coding single-stranded RNAs, regulate post-transcriptional cellular processes and participate in ALI/ARDS pathophysiology. Studies reveal that exosomes transport proteins, nucleic acids, and miRNAs to recipient cells, mediating intercellular communication. In ALI/ARDS models, exosomal miRNAs delivered to alveolar epithelial cells, endothelial cells, macrophages, and neutrophils critically modulate autophagy, pyroptosis, apoptosis, proliferation, inflammatory signaling, macrophage polarization, and neutrophil activation, either exacerbating or alleviating disease progression. Recent advances in engineering techniques have enhanced the therapeutic potential of exosomal miRNAs by overcoming limitations of natural exosomes. This review focuses on exosomal miRNA-mediated regulation of ALI/ARDS pathogenesis across key cell types, providing insights for novel therapeutic strategies.

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来源期刊
Zhonghua wei zhong bing ji jiu yi xue
Zhonghua wei zhong bing ji jiu yi xue Medicine-Critical Care and Intensive Care Medicine
CiteScore
1.00
自引率
0.00%
发文量
42
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