放射治疗期间抑制TGFβ增强免疫细胞浸润,减少尤文氏肉瘤转移。

IF 3.3 Q3 ONCOLOGY
Jessica D Daley, Elina Mukherjee, David Ferraro, A Carolina Tufino, Nathanael Bailey, Shanthi Bhaskar, Nivitha Periyapatna, Ian MacFawn, Sean Hartwick, Sheryl Kunning, Cynthia Hinck, Tullia C Bruno, Adam C Olson, Linda M McAllister-Lucas, Andrew P Hinck, Kristine Cooper, Riyue Bao, Anthony R Cillo, Kelly M Bailey
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引用次数: 0

摘要

尤文氏肉瘤(EwS)是一种侵袭性癌症,多发于青少年和年轻人。TGFβ的抑制作用正在针对复发性EwS的有限临床试验中进行测试。TGFβ是一种免疫抑制细胞因子,存在于潜伏和活性状态。TGFβ抑制对Ewing肿瘤微环境(TME)和Ewing肿瘤行为的功能影响在很大程度上仍然未知。在这里,我们使用人类Ewing肿瘤的单细胞RNAseq分析来证明免疫细胞是人类Ewing TME中TGFB1表达的最大贡献者。我们利用人源化小鼠EwS模型来证明这些模型中的TME特征与免疫缺陷小鼠的EwS肿瘤有显著差异。使用这种人源化模型,我们研究了放射治疗期间TGFβ抑制对EwS TME的影响,放射治疗通常用于治疗不可切除,转移性和复发/难治性EwS,已知可增强多种癌症中TGFβ的激活。利用三价配体TGFβ陷阱抑制TGF,我们证明TGFβ抑制与放射治疗联合使用,既增加了EwS免疫细胞浸润,又减少了体内肺转移负担。这些数据证明了免疫活性模型在解决EwS免疫生物学临床前问题方面的价值,并证明放射治疗期间抑制TGFβ是增强抗肿瘤免疫反应和提高转移性EwS治疗效果的有希望的策略。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
TGFβ inhibition during radiation therapy enhances immune cell infiltration and decreases metastases in Ewing sarcoma.

Ewing sarcoma (EwS) is an aggressive cancer diagnosed in adolescents and young adults. Inhibition of TGFβ is being tested in limited clinical trials for relapsed EwS. TGFβ is an immunosuppressive cytokine that exists in latent and active states. The functional impact of TGFβ inhibition on the Ewing tumor microenvironment (TME) and on Ewing tumor behavior remains largely unknown. Here, we use single-cell RNAseq analysis of human Ewing tumors to demonstrate that immune cells are the largest contributors of TGFB1 expression in the human Ewing TME. We utilize a humanized mouse model of EwS to demonstrate that TME signatures in these models differ significantly from EwS tumors developed in immunodeficient mice. Using this humanized model, we investigate the effect of TGFβ inhibition on the EwS TME during radiation therapy, a treatment that is commonly used to treat unresectable, metastatic, and relapsed/refractory EwS that is known to enhance TGFβ activation in multiple cancers. Utilizing a trivalent ligand TGFβ trap to inhibit TGF, we demonstrate that in combination with radiation therapy, TGFβ inhibition both increases EwS immune cell infiltration and decreases lung metastatic burden in vivo. These data demonstrate the value of immunocompetent models to address immune-biologic preclinical questions in EwS and demonstrate that TGFβ inhibition during radiation therapy is a promising strategy to enhance anti-tumor immune response and improve treatment efficacy for metastatic EwS.

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