脑膜瘤的癫痫发生:理论、推定的生物标志物和术后风险。

IF 6.6 1区 医学 Q1 CLINICAL NEUROLOGY
Epilepsia Pub Date : 2025-07-29 DOI:10.1111/epi.18559
William H Cook, Conor S Gillespie, Ali Bakhsh, Anthony G Marson, Michael D Jenkinson, Adel E Helmy
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引用次数: 0

摘要

颅脑膜瘤是最常见的原发性脑肿瘤类型,局灶性发病,肿瘤相关癫痫发作影响相当大比例的患者。30%有症状的术前患者和12%的术后患者会发生癫痫发作。虽然大多数患者可以通过手术治愈他们的肿瘤疾病,但癫痫发作会导致残疾,降低生活质量,延迟恢复驾驶和工作,并增加猝死的风险。肿瘤相关的癫痫发作也更有可能对抗癫痫药物(asm)产生耐药性。asm仅限于治疗癫痫发作的症状,但对引起或维持癫痫易感性的机制没有改善疾病的作用。有必要能够预测谁有发生术后癫痫发作的风险,以便有针对性地预防或更密切地监测那些风险较大的人。脑肿瘤相关癫痫发作的机制很可能是多因素的,与形态学、生化和代谢原因有关。脑肿瘤可能由于肿块效应、脑侵犯和瘤周脑水肿引起的刺激而引起皮质亢奋。炎症介质在动物模型和人类癫痫发作综合征中参与癫痫发生,并且有实验数据支持炎症介质作为癫痫发生的生物标志物的发展。脑膜瘤相关的癫痫发作尚不完全清楚,因此以目前的知识无法预测。在这篇综述中,我们讨论了脑肿瘤中癫痫发生的机制以及脑膜瘤相关癫痫发作的神经炎症机制。最后,我们评估脑膜瘤中癫痫发生的神经炎症生物标志物的潜力,并根据目前的文献推断当前的挑战,这些文献主要考虑癫痫和内在脑肿瘤。一项前瞻性随机对照试验(STOP’em: ISRCTN14381346)在英国开放,将确定两周预防性左乙西坦在seizure-naïve脑膜瘤手术患者中的作用,并提供获得患者连续血液测量的机会,以协助发现生物标志物。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Epileptogenesis in meningioma: Theories, putative biomarkers, and postoperative risk.

Cranial meningioma are the most common type of primary brain tumor, and focal onset, tumor-related seizures affect a significant proportion of patients. Seizures affect 30% of symptomatic preoperative patients and a further 12% of postoperative patients. Although most patients may be cured of their oncological disease by surgery, seizures confer disability, reduced quality-of-life, delayed return to driving and work, and increase the risk of sudden death. Tumor-associated seizures are also more likely to be resistant to antiseizure medications (ASMs). ASMs are limited to treating the symptoms of epilepsy-seizures-but have no disease-modifying effect on the mechanisms that cause or maintain seizure susceptibility. There is a need to be able to predict who is at risk of developing postoperative seizures for targeted prevention or closer monitoring of those at greater risk. Mechanisms underpinning brain tumor-related seizures are most likely multifactorial and related to morphological, biochemical, and metabolic causes. Brain tumors likely cause cortical hyperexcitability due to irritation caused by mass effect, brain invasion, and peritumoral brain edema. Inflammatory mediators are involved in epileptogenesis in animal models and human seizure syndromes and there are experimental data to support the development of inflammatory mediators as biomarkers for epileptogenesis. Meningioma-associated seizures are incompletely understood and consequently unpredictable with current knowledge. In this review, we discuss the proposed mechanisms of epileptogenesis in brain tumors and putative neuroinflammatory mechanisms for meningioma-associated seizures. Ultimately, we evaluate the potential of neuroinflammatory biomarkers of epileptogenesis in meningioma and the current challenges with extrapolating from current literature, which primarily consider epilepsy and intrinsic brain tumors. A prospective randomized controlled trial (STOP'EM: ISRCTN14381346) is open in the UK and will determine the role of two weeks of prophylactic levetiracetam in seizure-naïve patients undergoing meningioma surgery and provide an opportunity to obtain serial blood measurements from patients to assist with biomarker discovery.

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来源期刊
Epilepsia
Epilepsia 医学-临床神经学
CiteScore
10.90
自引率
10.70%
发文量
319
审稿时长
2-4 weeks
期刊介绍: Epilepsia is the leading, authoritative source for innovative clinical and basic science research for all aspects of epilepsy and seizures. In addition, Epilepsia publishes critical reviews, opinion pieces, and guidelines that foster understanding and aim to improve the diagnosis and treatment of people with seizures and epilepsy.
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