“通过激活nrf -2介导的抗氧化反应抑制心肌肥厚”的勘误表

IF 3.4 4区 医学 Q2 CARDIAC & CARDIOVASCULAR SYSTEMS
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引用次数: 0

摘要

史旭,张斌,褚忠等,“Wogonin通过激活nrf -2介导的抗氧化反应抑制心肌肥厚”,Cardiovascular Therapeutics 2021 (2021): 9995342, https://doi.org/10.1155/2021/9995342In文章中,由于稿件准备过程中图像选择错误,图4e中出现了错误。正确的图4如下所示:作者为这个错误道歉。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Corrigendum to “Wogonin Inhibits Cardiac Hypertrophy by Activating Nrf-2-Mediated Antioxidant Responses”

Corrigendum to “Wogonin Inhibits Cardiac Hypertrophy by Activating Nrf-2-Mediated Antioxidant Responses”

X. Shi, B. Zhang, Z. Chu, et al., “Wogonin Inhibits Cardiac Hypertrophy by Activating Nrf-2-Mediated Antioxidant Responses”, Cardiovascular Therapeutics 2021 (2021): 9995342, https://doi.org/10.1155/2021/9995342

In the article, there is an error in Figure 4e due to the incorrect selection of images during manuscript preparation. The correct Figure 4 is shown below:

The authors apologize for this error.

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来源期刊
Cardiovascular Therapeutics
Cardiovascular Therapeutics 医学-心血管系统
CiteScore
5.60
自引率
0.00%
发文量
55
审稿时长
6 months
期刊介绍: Cardiovascular Therapeutics (formerly Cardiovascular Drug Reviews) is a peer-reviewed, Open Access journal that publishes original research and review articles focusing on cardiovascular and clinical pharmacology, as well as clinical trials of new cardiovascular therapies. Articles on translational research, pharmacogenomics and personalized medicine, device, gene and cell therapies, and pharmacoepidemiology are also encouraged. Subject areas include (but are by no means limited to): Acute coronary syndrome Arrhythmias Atherosclerosis Basic cardiac electrophysiology Cardiac catheterization Cardiac remodeling Coagulation and thrombosis Diabetic cardiovascular disease Heart failure (systolic HF, HFrEF, diastolic HF, HFpEF) Hyperlipidemia Hypertension Ischemic heart disease Vascular biology Ventricular assist devices Molecular cardio-biology Myocardial regeneration Lipoprotein metabolism Radial artery access Percutaneous coronary intervention Transcatheter aortic and mitral valve replacement.
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