“P75NTR通过YAP-mTOR途径调节糖尿病小鼠的自噬,增加滤泡间表皮细胞的增殖,促进伤口愈合”的更正[BBA - Mol. Basis of Dis. 2024年3月;1870(3): 167012。

IF 4.2 2区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY
Zhenjie Wu , Chunyan Liu , Siyuan Yin , Ru Song , Jiaxu Ma , Rui Sun , Guoqi Cao , Yongpan Lu , Jian Liu , Linqi Su , Yibing Wang
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引用次数: 0

摘要

本文章由计算机程序翻译,如有差异,请以英文原文为准。
Corrigendum to “P75NTR regulates autophagy through the YAP-mTOR pathway to increase the proliferation of interfollicular epidermal cells and promote wound healing in diabetic mice” [Biochim. Biophys. Acta - Mol. Basis Dis.] 2024 Mar; 1870 (3): 167012
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来源期刊
CiteScore
12.30
自引率
0.00%
发文量
218
审稿时长
32 days
期刊介绍: BBA Molecular Basis of Disease addresses the biochemistry and molecular genetics of disease processes and models of human disease. This journal covers aspects of aging, cancer, metabolic-, neurological-, and immunological-based disease. Manuscripts focused on using animal models to elucidate biochemical and mechanistic insight in each of these conditions, are particularly encouraged. Manuscripts should emphasize the underlying mechanisms of disease pathways and provide novel contributions to the understanding and/or treatment of these disorders. Highly descriptive and method development submissions may be declined without full review. The submission of uninvited reviews to BBA - Molecular Basis of Disease is strongly discouraged, and any such uninvited review should be accompanied by a coverletter outlining the compelling reasons why the review should be considered.
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