结论：mettl3介导的TIM1通过igf2bp2依赖的方式促进巨噬细胞M1极化和炎症

IF 3.2 3区医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY

Journal of Biochemical and Molecular Toxicology Pub Date : 2025-07-16 DOI:10.1002/jbt.70396

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引用次数: 0

摘要

撤回：杜显荣，郭银光，赵小琴，张丽娟，范茹，李亚峰。mettl3介导的TIM1通过igf2bp2依赖的方式促进巨噬细胞M1极化和炎症，Journal of Biochemical and Molecular Toxicology 38, no. 5。10 (2024): e23845。上述文章于2024年9月12日在Wiley在线图书馆（wileyonlinelibrary.com）上发表，经作者、期刊主编Dr. Hari K. Bhat和Wiley期刊有限责任公司同意，该文章已被撤回，因为该文章未经所有相关贡献者的授权而发表。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

查看原文本刊更多论文

WITHDRAWAL: METTL3-Mediated TIM1 Promotes Macrophage M1 Polarization and Inflammation Through IGF2BP2-Dependent Manner

WITHDRAWAL: Xianrong Du, Yinguang Guo, Xiaoqin Zhao, Lijuan Zhang, Ru Fan, Yafeng Li. METTL3-mediated TIM1 promotes macrophage M1 polarization and inflammation through IGF2BP2-dependent manner, Journal of Biochemical and Molecular Toxicology 38, no. 10 (2024): e23845. https://doi.org/10.1002/jbt.23845.

The above article, published online on 12 September 2024, on Wiley Online Library (wileyonlinelibrary.com), has been withdrawn by agreement between the authors, the journal Editor-in-Chief, Dr. Hari K. Bhat, and Wiley Periodicals LLC. The withdrawal has been agreed because the article was published without authorisation of all relevant contributors.

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来源期刊

Journal of Biochemical and Molecular Toxicology 生物-毒理学

CiteScore

5.80

自引率

2.80%

发文量

277

审稿时长

6-12 weeks

期刊介绍： The Journal of Biochemical and Molecular Toxicology is an international journal that contains original research papers, rapid communications, mini-reviews, and book reviews, all focusing on the molecular mechanisms of action and detoxication of exogenous and endogenous chemicals and toxic agents. The scope includes effects on the organism at all stages of development, on organ systems, tissues, and cells as well as on enzymes, receptors, hormones, and genes. The biochemical and molecular aspects of uptake, transport, storage, excretion, lactivation and detoxication of drugs, agricultural, industrial and environmental chemicals, natural products and food additives are all subjects suitable for publication. Of particular interest are aspects of molecular biology related to biochemical toxicology. These include studies of the expression of genes related to detoxication and activation enzymes, toxicants with modes of action involving effects on nucleic acids, gene expression and protein synthesis, and the toxicity of products derived from biotechnology.