Carlos Ventura, Xiaowei Bogetti, Ji Young Lee, Ivet Bahar
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Allosteric Autoregulation of Ferroptosis Suppressor Protein 1 Activity by its N-myristoylated Tail.
Ferroptosis is a form of cell death characterized by iron-dependent accumulation of lipid peroxides. Ferroptosis suppressor protein 1 (FSP1) has been shown to work with glutathione peroxidase 4 (GPX4) to suppress ferroptosis through different antioxidant pathways. Many studies have been conducted on FSP1 to better understand its function and mechanism of action, which remained inconclusive in the absence of structural information on FSP1. Recent elucidation of FSP1 structures in different forms and advances in computational characterization of functional changes in its conformation provide us with the opportunity of dissecting FSP1 mechanism of action and gaining insights into critical sites and interactions that control its activity. We present the results from elastic network model analyses of cooperative changes in FSP1 structure, as well as those from molecular dynamics simulations of its interactions with the lipid bilayer and small molecules, toward assisting in future development of modulators of ferroptosis targeting FSP1. Our study reveals the critical role of N-terminal myristoylated tail in modulating the accessibility of the ligand-binding sites and in anchoring FSP1 to the membrane, giving insights into mechanisms of regulating FSP1 function.
期刊介绍:
Journal of Molecular Biology (JMB) provides high quality, comprehensive and broad coverage in all areas of molecular biology. The journal publishes original scientific research papers that provide mechanistic and functional insights and report a significant advance to the field. The journal encourages the submission of multidisciplinary studies that use complementary experimental and computational approaches to address challenging biological questions.
Research areas include but are not limited to: Biomolecular interactions, signaling networks, systems biology; Cell cycle, cell growth, cell differentiation; Cell death, autophagy; Cell signaling and regulation; Chemical biology; Computational biology, in combination with experimental studies; DNA replication, repair, and recombination; Development, regenerative biology, mechanistic and functional studies of stem cells; Epigenetics, chromatin structure and function; Gene expression; Membrane processes, cell surface proteins and cell-cell interactions; Methodological advances, both experimental and theoretical, including databases; Microbiology, virology, and interactions with the host or environment; Microbiota mechanistic and functional studies; Nuclear organization; Post-translational modifications, proteomics; Processing and function of biologically important macromolecules and complexes; Molecular basis of disease; RNA processing, structure and functions of non-coding RNAs, transcription; Sorting, spatiotemporal organization, trafficking; Structural biology; Synthetic biology; Translation, protein folding, chaperones, protein degradation and quality control.