{"title":"轻度智力迟钝遗传学的传统观点与新观点。","authors":"H O Akesson","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>Normal intelligence and mild mental retardation are generally assumed to be determined by additive polygenes. This old and basic theory is challenged by findings from modern population studies. The discrepancies concern frequency of mild mental retardation, sex ratio, and additional somatic handicaps in populations with mild mental retardation.</p>","PeriodicalId":76782,"journal":{"name":"Upsala journal of medical sciences. Supplement","volume":"44 ","pages":"30-3"},"PeriodicalIF":0.0000,"publicationDate":"1987-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Traditional views and new perspectives on the genetics of mild mental retardation.\",\"authors\":\"H O Akesson\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>Normal intelligence and mild mental retardation are generally assumed to be determined by additive polygenes. This old and basic theory is challenged by findings from modern population studies. The discrepancies concern frequency of mild mental retardation, sex ratio, and additional somatic handicaps in populations with mild mental retardation.</p>\",\"PeriodicalId\":76782,\"journal\":{\"name\":\"Upsala journal of medical sciences. Supplement\",\"volume\":\"44 \",\"pages\":\"30-3\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1987-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Upsala journal of medical sciences. Supplement\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Upsala journal of medical sciences. Supplement","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Traditional views and new perspectives on the genetics of mild mental retardation.
Normal intelligence and mild mental retardation are generally assumed to be determined by additive polygenes. This old and basic theory is challenged by findings from modern population studies. The discrepancies concern frequency of mild mental retardation, sex ratio, and additional somatic handicaps in populations with mild mental retardation.
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