MeCP2和PTEN基因在特异性学习障碍分子基础中的表达改变。

IF 2.8 4区 医学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
Fatma Atasever, Nil Özbilüm Şahin, Cansu Mercan Işık
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引用次数: 0

摘要

特殊学习障碍(SLD)是一种影响认知能力的神经发育障碍,如阅读、写作和数学。尽管遗传和表观遗传因素被认为起着重要作用,但SLD的分子机制仍不清楚。MeCP2是一种结合甲基化DNA的表观遗传调节剂,在神经元细胞的基因表达和SP调控中起着至关重要的作用。PTEN是一种肿瘤抑制基因,调节细胞生长、存活和凋亡,对维持突触完整性至关重要。在这项研究中,我们旨在检测MeCP2和PTEN在SLD患者中的表达。从血样中分离RNA,采用定量PCR (qPCR)检测基因表达。共有38名SLD患者和35名健康对照者参与了这项研究。我们的研究结果显示,与对照组相比,SLD组MeCP2上调15.44倍,PTEN下调13.66倍,表明基因表达平衡被破坏。重度SLD组与非重度SLD组间基因表达差异无统计学意义。这些发现提示MeCP2和PTEN的失调可能参与了SLD的病理生理,影响SP和神经元功能。总之,这些基因在SLD个体中的表达改变突出了早期诊断和治疗靶点的潜在生物标志物,为未来的研究和干预策略开辟了道路。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Altered Expression of MeCP2 and PTEN Genes in the Molecular Basis of Specific Learning Disorder.

Specific learning disorders (SLD) are neurodevelopmental disorders that affect cognitive abilities such as reading, writing, and mathematics. The molecular mechanisms underlying SLD remain unclear, though genetic and epigenetic factors are thought to play a significant role. MeCP2 is an epigenetic regulator that binds to methylated DNA, playing a crucial role in the regulation of gene expression and SP in neuronal cells. PTEN, a tumor suppressor gene, regulates cell growth, survival, and apoptosis, and is critical for maintaining synaptic integrity. In this study, we aimed to examine the expression of MeCP2 and PTEN in individuals with SLD. RNA was isolated from blood samples, and gene expression was assessed using quantitative PCR (qPCR). A total of 38 participants with SLD and 35 healthy controls were included in the study. Our results revealed a 15.44-fold upregulation of MeCP2 and a 13.66-fold downregulation of PTEN in the SLD group compared to controls, suggesting a disrupted balance of gene expression. There was no significant difference in gene expression between severe and non-severe SLD groups. These findings suggest that the dysregulation of MeCP2 and PTEN may be involved in the pathophysiology of SLD, influencing SP and neuronal function. In conclusion, the altered expression of these genes in individuals with SLD highlights potential biomarkers for early diagnosis and therapeutic targets, opening avenues for future research and intervention strategies.

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来源期刊
Journal of Molecular Neuroscience
Journal of Molecular Neuroscience 医学-神经科学
CiteScore
6.60
自引率
3.20%
发文量
142
审稿时长
1 months
期刊介绍: The Journal of Molecular Neuroscience is committed to the rapid publication of original findings that increase our understanding of the molecular structure, function, and development of the nervous system. The criteria for acceptance of manuscripts will be scientific excellence, originality, and relevance to the field of molecular neuroscience. Manuscripts with clinical relevance are especially encouraged since the journal seeks to provide a means for accelerating the progression of basic research findings toward clinical utilization. All experiments described in the Journal of Molecular Neuroscience that involve the use of animal or human subjects must have been approved by the appropriate institutional review committee and conform to accepted ethical standards.
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