昆虫共生病毒通过抑制E3泛素连接酶Sina促进植物虫媒病毒的传播。

IF 5.5 1区 医学 Q1 MICROBIOLOGY
PLoS Pathogens Pub Date : 2025-05-29 eCollection Date: 2025-05-01 DOI:10.1371/journal.ppat.1013178
Hui Wang, Jieting Zhang, Runfa Liu, You Li, Yu Du, Taiyun Wei
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引用次数: 0

摘要

共生病毒和虫媒病毒在昆虫媒介中具有协同作用的共同感染在自然界中很常见,但其潜在机制尚不清楚。本研究鉴定了一种新的共生病毒——叶蝉背背虫布雅病毒(RdBV),该病毒可提高水稻条纹花叶病毒(RSMV,植物横纹病病毒)在田间的传播效率。RSMV感染激活dorsalis E3泛素连接酶7 in absentia (RdSina)的表达,而RdBV感染抑制其表达。我们发现,RdSina直接靶向并介导RSMV磷酸化蛋白(P)的降解,从而减弱P诱导的病毒质形成,这对病毒复制至关重要。RdSina与RdBV的非结构蛋白NSs2相互作用,但不介导其泛素化。然而,NSs2与RSMV P竞争与RdSina结合,从而中和RdSina介导P降解的能力。此外,我们发现MYC转录因子与RdSina的启动子序列结合,激活其转录。然而,NSs2也直接结合RdSina的相同启动子序列,并竞争性地抑制myc激活的RdSina转录。总之,NSs2阻断了RdSina介导P降解的功能,最终促进了RSMV在共感染载体中的传播。这些发现阐明了昆虫共生病毒如何负调控E3泛素连接酶,从而有利于虫媒病毒通过共感染的昆虫媒介传播,这可能是自然界中普遍存在的现象。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
An insect symbiotic virus promotes the transmission of a phytoarbovirus via inhibiting E3 ubiquitin ligase Sina.

Co-infection with symbiotic viruses and arboviruses with synergistic effects in insect vectors are common in nature, but the underlying mechanism remains elusive. Here, we identify a novel symbiotic virus, leafhopper Recilia dorsalis bunyavirus (RdBV), which enhances the transmission efficiency of cytorhabdovirus rice stripe mosaic virus (RSMV, a plant rhabdovirus) in field. RSMV infection activates the expression of R. dorsalis E3 ubiquitin ligase Seven in absentia (RdSina), while RdBV infection suppresses its expression. We show that RdSina directly targets and mediates the degradation of RSMV phosphoprotein (P), thereby attenuating the formation of P-induced viroplasm that are crucial for viral replication. RdSina interacts with nonstructural protein NSs2 of RdBV but does not mediate its ubiquitination. However, NSs2 competes with RSMV P for binding to RdSina, thus neutralizing RdSina's ability in mediating P degradation. Furthermore, we find that the MYC transcription factor binds to the promoter sequences of RdSina, activating its transcription. However, NSs2 also directly binds to the same promoter sequences of RdSina and competitively suppresses MYC-activated RdSina transcription. Together, NSs2 obstructs the function of RdSina in mediating P degradation, ultimately promoting RSMV propagation in co-infected vectors. These findings elucidate how insect symbiotic viruses negatively regulate E3 ubiquitin ligases to benefit arbovirus transmission by co-infected insect vectors, which potentially is a common phenomenon in nature.

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来源期刊
PLoS Pathogens
PLoS Pathogens MICROBIOLOGY-PARASITOLOGY
自引率
3.00%
发文量
598
期刊介绍: Bacteria, fungi, parasites, prions and viruses cause a plethora of diseases that have important medical, agricultural, and economic consequences. Moreover, the study of microbes continues to provide novel insights into such fundamental processes as the molecular basis of cellular and organismal function.
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