[SGLT2抑制剂诱导的红细胞增多]。

Yoko Edahiro
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引用次数: 0

摘要

钠-葡萄糖共转运体(SGLT) 2抑制剂已被证明通过抑制葡萄糖在肾脏的重吸收和促进其排泄到尿液中来诱导降糖作用。SGLT2抑制剂还有助于减少心力衰竭和预防肾脏疾病进展,因此除了糖尿病患者外,这些药物在心力衰竭和慢性肾脏疾病患者中的应用越来越有吸引力。近年来出现了一个新的观察,SGLT2抑制剂诱导红细胞增殖,这一现象在红细胞增多症的鉴别诊断中值得考虑。解释红细胞增多机制的主流假说是由于SGLT2抑制剂的利尿作用导致的体液体积减少和血药浓度升高。然而,一种涉及促红细胞生成素产生增加的新机制也被假设。一项针对SGLT2抑制剂引起的红细胞增多的患者的研究报告指出,除了静脉切开术外,应慎重考虑停用SGLT2抑制剂治疗红细胞增多。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Erythrocytosis induced by SGLT2 inhibitors].

Sodium-glucose cotransporter (SGLT) 2 inhibitors have been demonstrated to induce hypoglycemic effects by inhibiting glucose reabsorption in the kidneys and promoting its excretion into urine. SGLT2 inhibitors also contribute to a reduction in heart failure and prevention of kidney disease progression, thus making these drugs increasingly attractive for use in patients with heart failure and chronic kidney disease in addition to patients with diabetes. A novel observation has emerged in recent years that SGLT2 inhibitors induce erythrocyte proliferation, a phenomenon that warrants consideration in the differential diagnosis of erythrocytosis. The prevailing hypothesis explaining the mechanism of erythrocytosis has been the decrease in fluid volume and increase in concentration of the blood which is attributable to the diuretic effect of SGLT2 inhibitors. However, a novel mechanism involving increased production of erythropoietin has also been postulated. A study of patients with erythrocytosis caused by SGLT2 inhibitors reported that careful consideration should be given to discontinuing SGLT2 inhibitors for erythrocytosis in addition to phlebotomy.

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