蛋白磷酸酶1调控亚基PNUTS防止CENP-A错位和染色体不稳定性。

IF 2.7 2区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY
Molecular and Cellular Biology Pub Date : 2025-01-01 Epub Date: 2025-04-24 DOI:10.1080/10985549.2025.2487010
Vinutha Balachandra, Makenzie Thomas, Roshan L Shrestha, Subhash Chandra Sethi, Raj Chari, Shinjen Lin, Ken Chih-Chien Cheng, Tatiana S Karpova, Natasha J Caplen, Munira A Basrai
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引用次数: 0

摘要

染色体不稳定性(CIN)是癌症的一个主要标志,可由着丝粒或着丝粒结构完整性的缺陷驱动。细胞分裂过程中磷酸化和去磷酸化活动的协调控制是确保染色体稳定性的关键。着丝粒组蛋白H3变异体CENP-A在许多癌症中都有过表达,它在非着丝粒区域的错误定位促进了CIN。我们在全基因组siRNA筛选中发现蛋白磷酸酶1 (PP1)核靶向亚基(PNUTS)是导致核CENP-A水平升高的基因缺失的首选候选基因。在这里,我们定义了PNUTS在防止CENP-A错定位和CIN中的作用。PNUTS的耗竭导致在整个细胞周期中以pp1依赖的方式产生高核CENP-A水平。与这些结果一致,在pnuts缺失的细胞有丝分裂染色体上观察到CENP-A及其相互作用伙伴CENP-C的错误定位。在pnuts缺失的细胞中也观察到着丝点完整性和CIN表型的缺陷。在机制上,我们发现组蛋白H3.3伴侣DAXX的缺失抑制了pnuts缺失细胞中CENP-A的错误定位和微核发生率。总之,我们的研究强调了PNUTS介导的磷酸化调控在预防CENP-A错定位和CIN中的重要性。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Protein Phosphatase 1 Regulatory Subunit PNUTS Prevents CENP-A Mislocalization and Chromosomal Instability.

Chromosomal instability (CIN), a major hallmark of cancer, can be driven by defects in the integrity of centromere or kinetochore structure. Coordinated control of phosphorylation and dephosphorylation activities during cell division is critical to ensure chromosomal stability. Overexpression of the centromeric histone H3 variant CENP-A is observed in many cancers, and its mislocalization to noncentromeric regions promotes CIN. We identified protein phosphatase 1 (PP1) nuclear targeting subunit (PNUTS) as a top candidate in a genome-wide siRNA screen for gene depletions that lead to increased nuclear CENP-A levels. Here, we define a role for PNUTS in preventing CENP-A mislocalization and CIN. Depletion of PNUTS resulted in high nuclear CENP-A levels throughout the cell cycle in a PP1-dependent manner. Consistent with these results, mislocalization of CENP-A and its interacting partner CENP-C were observed on mitotic chromosomes from PNUTS-depleted cells. Defects in kinetochore integrity and CIN phenotypes were also observed in PNUTS-depleted cells. Mechanistically, we show that depletion of the histone H3.3 chaperone DAXX suppresses the mislocalization of CENP-A and micronuclei incidence in PNUTS-depleted cells. In summary, our studies highlight the importance of phospho-regulation mediated by PNUTS in preventing CENP-A mislocalization and CIN.

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来源期刊
Molecular and Cellular Biology
Molecular and Cellular Biology 生物-生化与分子生物学
CiteScore
9.80
自引率
1.90%
发文量
120
审稿时长
1 months
期刊介绍: Molecular and Cellular Biology (MCB) showcases significant discoveries in cellular morphology and function, genome organization, regulation of genetic expression, morphogenesis, and somatic cell genetics. The journal also examines viral systems, publishing papers that emphasize their impact on the cell.
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