精神分裂症和抑郁症共有的遗传和神经机制

IF 9.6 1区 医学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY
Yingying Xie, Jilian Fu, Liping Liu, Xijin Wang, Feng Liu, Meng Liang, Hesheng Liu, Wen Qin, Chunshui Yu
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引用次数: 0

摘要

精神分裂症(SCZ)和抑郁症是两种普遍存在的精神障碍,其特征是共病和症状重叠,但潜在的遗传和神经机制在很大程度上仍然难以捉摸。在这里,我们研究了欧洲人SCZ和抑郁症共有的遗传变异和神经影像学变化,然后将我们的研究扩展到跨祖先(欧洲人和东亚人)人群。通过条件分析和联合分析,我们发现欧洲人的SCZ和抑郁症共有213个遗传变异,其中82.6%在跨祖先人群中被复制。共享风险变异比随机变异表现出更高的有害程度,并且在突触相关功能中富集,其中不到3%的共享变异在两种疾病之间表现出水平多效性。孟德尔随机化分析表明,SCZ与抑郁症之间存在相互的因果关系。通过多性状遗传共定位分析,我们确定了SCZ和抑郁症共有的13种体积表型。特别值得注意的是左岛和极平面的体积减少,这是通过对先前研究的大规模荟萃分析和首次发作drug-naïve患者的独立神经影像学数据集进行验证的。这些发现表明,共同的遗传风险变异、突触功能障碍和大脑结构变化可能是SCZ和抑郁症共病和症状重叠的基础。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Genetic and neural mechanisms shared by schizophrenia and depression

Genetic and neural mechanisms shared by schizophrenia and depression

Schizophrenia (SCZ) and depression are two prevalent mental disorders characterized by comorbidity and overlapping symptoms, yet the underlying genetic and neural mechanisms remain largely elusive. Here, we investigated the genetic variants and neuroimaging changes shared by SCZ and depression in Europeans and then extended our investigation to cross-ancestry (Europeans and East Asians) populations. Using conditional and conjunctional analyses, we found 213 genetic variants shared by SCZ and depression in Europeans, of which 82.6% were replicated in the cross-ancestry population. The shared risk variants exhibited a higher degree of deleteriousness than random and were enriched for synapse-related functions, among which fewer than 3% of shared variants showed horizontal pleiotropy between the two disorders. Mendelian randomization analyses indicated reciprocal causal effects between SCZ and depression. Using multiple trait genetic colocalization analyses, we pinpointed 13 volume phenotypes shared by SCZ and depression. Particularly noteworthy were the shared volume reductions in the left insula and planum polare, which were validated through large-scale meta-analyses of previous studies and independent neuroimaging datasets of first-episode drug-naïve patients. These findings suggest that the shared genetic risk variants, synapse dysfunction, and brain structural changes may underlie the comorbidity and symptom overlap between SCZ and depression.

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来源期刊
Molecular Psychiatry
Molecular Psychiatry 医学-精神病学
CiteScore
20.50
自引率
4.50%
发文量
459
审稿时长
4-8 weeks
期刊介绍: Molecular Psychiatry focuses on publishing research that aims to uncover the biological mechanisms behind psychiatric disorders and their treatment. The journal emphasizes studies that bridge pre-clinical and clinical research, covering cellular, molecular, integrative, clinical, imaging, and psychopharmacology levels.
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