当手术不是罪魁祸首:一例钡中毒被误认为是术后并发症

Maria Mourão , Raquel Lalanda , Vítor Correia , Filipa Nogueira , Pedro Marques , Olavo Gomes
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引用次数: 0

摘要

钡中毒可从胃肠运动亢进(呕吐、腹部痉挛和腹泻)发展到弛缓性麻痹、反射性松弛、呼吸衰竭和心血管功能障碍。本报告的目的是提出一个25岁的女性严重肥胖,谁接受了袖胃切除术并发缝合线泄漏和随后的胃狭窄。狭窄消退后,患者出现了无明显原因的反复胃肠道症状,并伴有低血钾。随着时间的推移,她出现了全身肌肉无力。患者以精神状态改变、眼球震颤、全身肌无力、低钾血症(2.7 mmol/L)就诊于急诊科。对神经系统症状进行了彻底的病因学调查。入院后12天检测显示尿钡水平升高(43.9 µg/L),提示入院时可识别出较高水平,可能导致呕吐和肌肉无力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
When surgery isn't the culprit: A case report of barium poisoning mistaken for a post-operative complication
Barium poisoning can progress from gastrointestinal hypermotility (vomiting, abdominal cramps, and diarrhea) to flaccid paralysis, areflexia, respiratory failure, and cardiovascular dysfunction. This report aims to present a case of a 25-year-old woman with severe obesity, who underwent a sleeve gastrectomy complicated by a suture line leak and subsequent gastric stenosis. Following the resolution of the stenosis, the patient experienced recurrent gastrointestinal symptoms without an apparent cause, accompanied by hypokalemia. Over time, she developed generalized muscle weakness. The patient presented to the ED with altered mental status, nystagmus, generalized muscular weakness, and hypokalemia (2.7 mmol/L). A thorough etiological investigation of neurological symptoms was initiated. Testing revealed elevated urinary barium levels (43.9 µg/L) 12 days after admission, suggesting that higher levels would have been identifiable at admission and may have contributed to vomiting and muscular weakness.
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