[CpG交通灯参与活性DNA去甲基化]。

Q3 Medicine
A V Lioznova, Yu A Medvedeva
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引用次数: 0

摘要

DNA甲基化是密切参与基因表达表观遗传调控的重要机制之一。然而,DNA甲基化与表达之间的关系尚不完全清楚。据报道,DNA甲基化变化是基因表达的原因,反之亦然——基因表达变化导致甲基化变化的例子。此前,我们引入了CpG红绿灯的概念——CpG位点的甲基化水平与附近基因的表达显著相关——并展示了它们在增强子调控中的重要作用。在这项工作中,我们表明CpG交通信号灯甲基化水平在细胞群体中是异质的,并表明这种特征是由它们的动态去甲基化引起的。CpG红绿灯中的5hmC富集和TET2定位位点证实了我们的假设。为了找出CpG位点的甲基化是基因表达的原因还是结果,我们应用了Jonas Peters及其合作者开发的因果方向方法的评估。我们确定,在预测甲基化变化是基因表达变化的原因的CpG位点(M→E CpG位点),甲基化水平在不同细胞中更稳定,活性去甲基化发生的频率低于CpG位点(可能是基因表达变化导致甲基化变化的CpG位点(E→M CpG位点)。我们还发现,与E→M CpG位点更常见的基因体相比,启动子区域的M→E CpG位点的份额更大。基于这些观察结果,我们认为M→E CpG位点的甲基化是稳定的,并起着“开/关”开关的作用。相反,E→M位点的甲基化是动态的,在细胞群中不同细胞之间是可变的,并且主要是由活性去甲基化引起的。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[CpG Traffic Lights Are Involved in Active DNA Demethylation].

DNA methylation is one of the most important mechanisms closely involved in the epigenetic regulation of gene expression. However, the relationship between DNA methylation and expression is not completely understood. There are reported examples of changes in DNA methylation being the cause of gene expression, and vice versa - examples of changes in gene expression to entail changes in methylation. Earlier, we introduced the concept of CpG traffic lights - individual CpG sites methylation levels of which significantly correlate with the expression of a gene nearby - and showed their important role in the regulation of enhancers. In this work, we show that the levels of CpG traffic lights methylation are heterogeneous in the cell population, and suggest that this feature is caused by their dynamic demethylation. The 5hmC enrichment and TET2 localization sites in CpG traffic lights confirm our hypothesis. In order to find out whether methylation of CpG sites is the cause or the consequence of the gene expression, we applied the assessment of the causation direction method developed by Jonas Peters and co-authors. We determined that in CpG sites where changes in methylation are predicted to be the cause of gene expression changes (M→E CpG sites), methylation levels are more stable in different cells and active demethylation occurs less often than in CpG sites where presumably changes in gene expression cause changes in methylation (E→M CpG sites). We also show a greater share of M→E CpG sites in the promoter regions than in the bodies of the genes where E→M CpG sites are more common. Based on these observations, we believe that methylation of M→E CpG sites is stable and functions as an "on/off" switch. On the contrary methylation of E→M sites is dynamic, variable between cells in a cell population, and is caused primarily by active demethylation.

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来源期刊
Molekulyarnaya Biologiya
Molekulyarnaya Biologiya Medicine-Medicine (all)
CiteScore
0.70
自引率
0.00%
发文量
131
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