IF 3.2 3区 医学 Q2 PHARMACOLOGY & PHARMACY
Molecular Pharmacology Pub Date : 2025-01-01 Epub Date: 2024-11-29 DOI:10.1016/j.molpha.2024.100006
Gavin M Traber, Mei-Juan Tu, Su Guan, Neelu Batra, Ai-Ming Yu
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引用次数: 0

摘要

重新引入在非小细胞肺癌(NSCLC)中消耗殆尽的抑制肿瘤的微RNA-7-5p(miR-7)是一种新的治疗方法,而以前的研究主要使用在体外化疗的miR-7模拟物。在这里,我们旨在确定在体内产生的带有有效载荷 miR-7 的新型生物工程 RNA(BioRNA/miR-7)分子的药理作用和治疗潜力。首先,通过共聚焦成像和免疫印迹研究,我们发现 BioRNA/miR-7 改变了 NSCLC 细胞线粒体的形态,同时下调了已知的靶基因:表皮生长因子受体(EGFR)、线粒体溶质载体家族 25A37 (SLC25A37) 和导入内膜转运酶亚基 (TIM50)。其次,通过荧光素酶报告和免疫印迹研究,我们验证了线粒体酰甘油激酶(AGK)是 miR-7 的一个新的直接靶点。第三,通过实时活细胞分析,我们发现 BioRNA/miR-7 可调节线粒体呼吸和糖酵解能力。第四,活细胞和终点存活率研究表明,BioRNA/miR-7 与培美曲塞(PEM)联用可产生抑制 NSCLC 细胞生长的强大协同效应,这与细胞内 PEM 积累增加有关,液相色谱串联质谱法可对 PEM 进行定量。最后,通过使用 NSCLC 患者异种移植小鼠模型进行体内治疗研究,我们证明了 BioRNA/miR-7 单药治疗和与 PEM 联合治疗在控制肿瘤进展方面的有效性和耐受性。我们的研究成果确立了 miR-7 在 NSCLC 代谢和 PEM 处置中的作用,并支持我们在体内生产的新型 BioRNA/miR-7-5p 用于分子药理学研究。我们的研究结果进一步说明了 BioRNA/miR-7 加 PEM 组合作为一种潜在治疗方法来对抗 NSCLC 肿瘤进展的潜力。意义声明:MiR-7是一种在非小细胞肺癌(NSCLC)中消耗殆尽的肿瘤抑制性微RNA,体外化学工程miR-7模拟物被证明可抑制NSCLC细胞衍生异种移植小鼠的肿瘤生长。在这里,一种新型体内生物工程 miR-7 分子(即 BioRNA/miR-7)被用来有效控制目标基因的表达和 NSCLC 细胞的新陈代谢。此外,BioRNA/miR-7还能显著提高培美曲塞在NSCLC患者肿瘤小鼠中的抗肿瘤活性,从而证明了miR-7在NSCLC代谢中的作用以及BioRNA/miR-7改善NSCLC治疗的潜力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Bioengineered miR-7-5p modulates non-small cell lung cancer cell metabolism to improve therapy.

Reintroduction of tumor-suppressive microRNA-7-5p (miR-7) that is depleted in non-small cell lung cancer (NSCLC) represents a new therapeutic approach, whereas previous studies mainly used miR-7 mimics chemoengineered in vitro. Here we aim to establish the pharmacological actions and therapeutic potential of novel bioengineered RNA bearing a payload miR-7 (BioRNA/miR-7) molecule produced in vivo. First, through confocal imaging and immunoblot studies, we revealed that BioRNA/miR-7 altered NSCLC cell mitochondrial morphology accompanied by the downregulation of known target genes, epidermal growth factor receptor (EGFR), mitochondrial solute carrier family 25A37 (SLC25A37), and import inner membrane translocase subunit (TIM50). Second, through luciferase reporter and immunoblot studies, we validated mitochondrial acylglycerol kinase (AGK) as a new direct target for miR-7. Third, through real-time live-cell analyses, we revealed BioRNA/miR-7 to modulate mitochondrial respiration and glycolytic capacity. Fourth, live-cell and endpoint viability studies demonstrated that the combination of BioRNA/miR-7 with pemetrexed (PEM) elicited a strong synergistic effect to inhibit NSCLC cell growth, associated with an increased intracellular PEM accumulation, as quantified by a liquid chromatography tandem mass spectrometry method. Finally, through in vivo therapy study using NSCLC patient-derived xenograft mouse model, we demonstrated the efficacy and tolerability of BioRNA/miR-7 monotherapy and combination therapy with PEM to control tumor progression. Our collective works establish a role for miR-7 in NSCLC metabolism and PEM disposition and support our novel, in vivo produced BioRNA/miR-7-5p for molecular pharmacological research. Our findings further illustrate the potential of BioRNA/miR-7 plus PEM combination as a potential treatment to combat NSCLC tumor progression. SIGNIFICANCE STATEMENT: MiR-7 is a tumor-suppressive microRNA depleted in non-small cell lung cancer (NSCLC), and in vitro chemoengineered miR-7 mimics were shown to inhibit tumor growth in NSCLC cell-derived xenograft mice. Here, a novel in vivo bioengineered miR-7 molecule, namely BioRNA/miR-7, was used to effectively control target gene expression and NSCLC cell metabolism. Furthermore, BioRNA/miR-7 was demonstrated to remarkably improve pemetrexed antitumor activity in NSCLC patient-derived tumor mice, supporting the role of miR-7 in NSCLC metabolism and potential for BioRNA/miR-7 to improve NSCLC therapy.

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来源期刊
Molecular Pharmacology
Molecular Pharmacology 医学-药学
CiteScore
7.20
自引率
2.80%
发文量
50
审稿时长
3-6 weeks
期刊介绍: Molecular Pharmacology publishes findings derived from the application of innovative structural biology, biochemistry, biophysics, physiology, genetics, and molecular biology to basic pharmacological problems that provide mechanistic insights that are broadly important for the fields of pharmacology and toxicology. Relevant topics include: Molecular Signaling / Mechanism of Drug Action Chemical Biology / Drug Discovery Structure of Drug-Receptor Complex Systems Analysis of Drug Action Drug Transport / Metabolism
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