pir-hsa-216911通过抑制TLR4启动的GSDMD激活来抑制肝癌细胞的焦亡。

IF 6.1 2区 生物学 Q1 CELL BIOLOGY
Zhouxiang Liao, Lichao Yang, Xiaojing Cheng, Xuejing Huang, Qi Zhang, Daoqiang Wen, Zhenyu Song, Yasi Li, Sha Wen, Yongfeng Li, Meizhen Ou, Zhangnan Huang, Tianqi Liu, Min He
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引用次数: 0

摘要

肝细胞癌(HCC)是一个全球性的健康问题,是全球癌症相关死亡的第四大原因。然而,piwi相互作用rna (pirna)在HCC过程中的作用尚未得到广泛探讨。通过小RNA测序,我们的研究发现了一种特异性的piRNA, pir-hsa-216911,它在HCC细胞中高表达。pir-hsa-216911的过表达促进HCC细胞侵袭并抑制细胞死亡,特别是焦亡。敲除pir-hsa-216911导致细胞焦亡活性增加,导致caspase-1和GSDMD的激活。进一步分析发现pir-hsa-216911靶向并抑制TLR4, TLR4是HCC中与焦亡相关的关键基因。在Huh7细胞系中,pir-hsa-216911敲除证实了其通过沉默TLR4来抑制TLR4/NFκB/NLRP3通路的作用。敲除pir-hsa-216911显著抑制Huh7异种移植瘤的形成。在HCC患者中,pir-hsa-216911在脂肪变性HCC肿瘤样品中高表达,抑制TLR4表达,抑制GSDMD激活。本研究将pir-hsa-216911作为HCC中新的高表达piRNA,通过沉默TLR4抑制GSDMD激活来抑制肝细胞凋亡。这些发现对HCC分子分型和癌症治疗的潜在靶点具有重要意义。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
pir-hsa-216911 inhibit pyroptosis in hepatocellular carcinoma by suppressing TLR4 initiated GSDMD activation.

Hepatocellular carcinoma (HCC) is a global health concern, ranking as the fourth leading cause of cancer-related deaths worldwide. However, the role of piwi-interacting RNAs (piRNAs) in HCC processes has not been extensively explored. Through small RNA sequencing, our study identified a specific piRNA, pir-hsa-216911, which is highly expressed in HCC cells. This overexpression of pir-hsa-216911 promotes HCC cell invasion and inhibits cell death, particularly pyroptosis. Knocking out pir-hsa-216911 led to increased cell pyroptosis activity, resulting in the activation of caspase-1 and GSDMD. Further analysis revealed that pir-hsa-216911 targets and suppresses TLR4, a key gene associated with pyroptosis in HCC. In the Huh7 cell line, pir-hsa-216911 knockout confirmed its role in suppressing the TLR4/NFκB/NLRP3 pathway by silencing TLR4. Knocking out pir-hsa-216911 significantly inhibited the formation of Huh7 xenograft tumor. In HCC patients, pir-hsa-216911 was highly expressed in HCC tumor samples with steatosis, suppressing TLR4 expression and inhibiting GSDMD activation. This study introduces pir-hsa-216911 as a new high-expressing piRNA in HCC, which inhibits pyroptosis by silencing TLR4 to suppress GSDMD activation. These findings have significant implications for HCC molecular subtyping and as a potential target for cancer therapy.

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来源期刊
Cell Death Discovery
Cell Death Discovery Biochemistry, Genetics and Molecular Biology-Cell Biology
CiteScore
8.30
自引率
1.40%
发文量
468
审稿时长
9 weeks
期刊介绍: Cell Death Discovery is a multidisciplinary, international, online-only, open access journal, dedicated to publishing research at the intersection of medicine with biochemistry, pharmacology, immunology, cell biology and cell death, provided it is scientifically sound. The unrestricted access to research findings in Cell Death Discovery will foster a dynamic and highly productive dialogue between basic scientists and clinicians, as well as researchers in industry with a focus on cancer, neurobiology and inflammation research. As an official journal of the Cell Death Differentiation Association (ADMC), Cell Death Discovery will build upon the success of Cell Death & Differentiation and Cell Death & Disease in publishing important peer-reviewed original research, timely reviews and editorial commentary. Cell Death Discovery is committed to increasing the reproducibility of research. To this end, in conjunction with its sister journals Cell Death & Differentiation and Cell Death & Disease, Cell Death Discovery provides a unique forum for scientists as well as clinicians and members of the pharmaceutical and biotechnical industry. It is committed to the rapid publication of high quality original papers that relate to these subjects, together with topical, usually solicited, reviews, editorial correspondence and occasional commentaries on controversial and scientifically informative issues.
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