端粒酶介导的抗衰老干预。

Q1 Biochemistry, Genetics and Molecular Biology
Phoebe L Dunn, Dhenugen Logeswaran, Julian J-L Chen
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引用次数: 0

摘要

衰老过程涉及到生物体一生中染色体完整性的逐渐下降。在真核生物中,端粒是覆盖在线性染色体末端的保护性dna -蛋白质复合物。端粒DNA由长段短的“TTAGGG”重复序列组成,在包括人类在内的大多数真核生物中都是保守的。由于传统的DNA聚合酶无法完全复制染色体末端,端粒会随着DNA的每一轮复制而逐渐缩短,这被称为“末端复制问题”。端粒酶通过在染色体末端重新添加端粒重复序列来抵消端粒DNA的损失。种系细胞和干细胞维持显著水平的端粒酶活性来维持端粒长度,并且几乎可以无限分裂。然而,干细胞的分化伴随着端粒酶基因表达的失活,导致体细胞在连续分裂过程中端粒逐渐缩短。极短的端粒引发并维持持续的DNA损伤反应,导致细胞永久生长停滞,称为细胞衰老,这是细胞衰老的标志。组织和器官中衰老细胞的积累有助于机体衰老。因此,预防端粒缩短是延缓甚至逆转细胞衰老的一种有希望的手段。在本章中,我们总结了通过增加端粒酶水平或活性来减缓端粒缩短的潜在抗衰老干预措施,并讨论了这些策略的风险、益处和未来前景。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Telomerase-Mediated Anti-Ageing Interventions.

The ageing process involves a gradual decline of chromosome integrity throughout an organism's lifespan. Telomeres are protective DNA-protein complexes that cap the ends of linear chromosomes in eukaryotic organisms. Telomeric DNA consists of long stretches of short "TTAGGG" repeats that are conserved across most eukaryotes including humans. Telomeres shorten progressively with each round of DNA replication due to the inability of conventional DNA polymerase to completely replicate the chromosome ends, known as the "end-replication problem". The telomerase enzyme counteracts the telomeric DNA loss by de novo addition of telomeric repeats onto chromosomal ends. Germline and stem cells maintain significant levels of telomerase activity to maintain telomere length and can divide almost indefinitely. However, the differentiation of stem cells accompanies the inactivation of telomerase gene expression, resulting in the progressive shortening of telomeres in somatic cells over successive divisions. Critically short telomeres elicit and sustain a persistent DNA damage response leading to permanent growth arrest of cells known as cellular senescence, a hallmark of cellular ageing. The accumulation of senescent cells in tissues and organs contributes to organismal ageing. Thus, the prevention of telomere shortening is a promising means to delay or even reverse cellular ageing. In this chapter, we summarize potential anti-ageing interventions that mitigate telomere shortening through increasing telomerase level or activity and discuss these strategies' risks, benefits, and future outlooks.

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来源期刊
Sub-cellular biochemistry
Sub-cellular biochemistry Biochemistry, Genetics and Molecular Biology-Biochemistry
CiteScore
5.90
自引率
0.00%
发文量
33
期刊介绍: The book series SUBCELLULAR BIOCHEMISTRY is a renowned and well recognized forum for disseminating advances of emerging topics in Cell Biology and related subjects. All volumes are edited by established scientists and the individual chapters are written by experts on the relevant topic. The individual chapters of each volume are fully citable and indexed in Medline/Pubmed to ensure maximum visibility of the work.
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