运动通过肌肽/Slc15a2-p53轴减轻放射后造血干细胞损伤。

IF 8.2 2区 生物学 Q1 CELL BIOLOGY
Hao Zeng, Naicheng Chen, Fang Chen, Xiaoyi Zhong, Lijing Yang, Yukai Lu, Mo Chen, Mingqiang Shen, Song Wang, Shilei Chen, Jia Cao, Xi Zhang, Jinghong Zhao, Yang Xu, Junping Wang, Mengjia Hu
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引用次数: 0

摘要

电离辐射(IR)可引起造血干细胞(hsc)的严重功能障碍,导致急性或长期的骨髓抑制。近年来,体育锻炼已被认为是一种健康的生活方式,因为它可以对抗各种疾病。然而,它是否提供对红外辐射的保护还不完全清楚。在这项研究中,我们揭示了长期适度运动通过产生骨骼肌肌肽来减轻ir诱导的造血损伤。我们发现运动小鼠在IR后HSC数量和功能的损失减少,同时骨髓损伤减轻。有趣的是,这些影响在很大程度上被骨骼肌中肌肽合成酶Carns1的特异性缺失所消除。相反,肌肽处理可以保护hsc免受ir诱导的损伤。在机制上,我们证明了运动产生的肌肽通过Slc15a2特异性转运到hsc,然后通过直接与其核心dna结合域相互作用抑制p53的转录活性,导致p53靶基因p21和Puma下调,从而促进辐照hsc的增殖和存活,抑制衰老。更重要的是,在人类脐带血来源的造血干细胞中观察到肌肽/Slc15a2-p53轴的类似作用。总的来说,我们的数据显示适度运动或补充肌肽可能是潜在的抗辐射策略。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Exercise alleviates hematopoietic stem cell injury following radiation via the carnosine/Slc15a2-p53 axis.

Ionizing radiation (IR) can cause severe dysfunction of hematopoietic stem cells (HSCs), leading to acute or prolonged myelosuppression. In recent years, physical exercise has been recognized as a healthy lifestyle as it can fight a variety of diseases. However, whether it provides protection against IR is not fully understood. In this study, we revealed that long-term moderate exercise mitigated IR-induced hematopoietic injury by generating carnosine from skeletal muscles. We found that exercised mice displayed reduced loss of HSC number and function after IR, accompanied by alleviated bone marrow damage. Interestingly, these effects were largely abrogated by specific deletion of carnosine synthase Carns1 in skeletal muscles. In contrast, carnosine treatment protected HSCs against IR-induced injury. Mechanistically, we demonstrated that exercise-generated carnosine was specifically transported to HSCs via Slc15a2 and then inhibited p53 transcriptional activity by directly interacting with its core DNA-binding domain, which led to downregulation of the p53 target genes p21 and Puma, thus promoting the proliferation and survival and inhibiting the senescence of irradiated HSCs. More importantly, a similar role of the carnosine/Slc15a2-p53 axis was observed in human cord blood-derived HSCs. Collectively, our data reveal that moderate exercise or carnosine supplementation may be potential antiradiation strategies.

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来源期刊
CiteScore
11.00
自引率
0.00%
发文量
180
期刊介绍: Cell Communication and Signaling (CCS) is a peer-reviewed, open-access scientific journal that focuses on cellular signaling pathways in both normal and pathological conditions. It publishes original research, reviews, and commentaries, welcoming studies that utilize molecular, morphological, biochemical, structural, and cell biology approaches. CCS also encourages interdisciplinary work and innovative models, including in silico, in vitro, and in vivo approaches, to facilitate investigations of cell signaling pathways, networks, and behavior. Starting from January 2019, CCS is proud to announce its affiliation with the International Cell Death Society. The journal now encourages submissions covering all aspects of cell death, including apoptotic and non-apoptotic mechanisms, cell death in model systems, autophagy, clearance of dying cells, and the immunological and pathological consequences of dying cells in the tissue microenvironment.
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