BMP-9通过TGF-β信号介导进行性骨化纤维发育不良的纤维增殖。

IF 9 1区 医学 Q1 MEDICINE, RESEARCH & EXPERIMENTAL
EMBO Molecular Medicine Pub Date : 2025-01-01 Epub Date: 2024-12-03 DOI:10.1038/s44321-024-00174-3
Chengzhu Zhao, Yoshiko Inada, Souta Motoike, Daisuke Kamiya, Kyosuke Hino, Makoto Ikeya
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引用次数: 0

摘要

进行性骨化纤维发育不良(FOP)是一种罕见的遗传性疾病,表现为软组织进行性异位骨化(HO)。早期FOP的特征是反复发作的疼痛组织肿胀(突然发作),随后有大量增殖激活的间充质间质细胞(MSCs)引起HO。然而,突发进展的机制仍不清楚。在这项研究中,我们评估了从FOP患者来源的诱导多能干细胞(top - ipscs)中获得的MSCs的增殖,以阐明突发事件的机制,并发现骨形态发生蛋白(BMP)-9通过异常激活转化生长因子(TGF)-β信号通路介导的MSCs增殖增强。在FOP模型小鼠中,升高的BMP-9水平与SMAD2/3磷酸化升高和受影响组织中细胞增殖增加相关,而全身BMP-9中和和敲除可减轻发作和HO。因此,BMP-9异常地转导TGF-β信号并诱导纤维增殖,引发急性发作。这项研究为未来FOP治疗的发展提供了新的见解。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
BMP-9 mediates fibroproliferation in fibrodysplasia ossificans progressiva through TGF-β signaling.

Fibrodysplasia ossificans progressiva (FOP) is a rare genetic disorder presenting with progressive heterotopic ossification (HO) in soft tissues. Early-stage FOP is characterized by recurrent episodes of painful tissue swelling (flare-ups), with numerous proliferation-activated mesenchymal stromal cells (MSCs) subsequently causing HO. However, the mechanisms underlying flare-up progression remain unclear. In this study, we evaluated the proliferation of MSCs obtained from FOP patient-derived induced pluripotent stem cells (FOP-iPSCs) to elucidate the mechanisms underlying flare-ups and found that bone morphogenetic protein (BMP)-9 mediated enhanced proliferation by abnormal activation of transforming growth factor (TGF)-β signaling pathway in MSCs from FOP-iPSCs. In FOP model mice, elevated BMP-9 levels correlated with elevated phosphorylation of SMAD2/3 and increased cellular proliferation in the affected tissues, while systemic BMP-9 neutralization and knockout mitigated flare-ups and HO. Thus, BMP-9 aberrantly transduces TGF-β signaling and induces fibroproliferation, initiating flare-ups. This study provides novel insights into the development of future FOP therapies.

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来源期刊
EMBO Molecular Medicine
EMBO Molecular Medicine 医学-医学:研究与实验
CiteScore
17.70
自引率
0.90%
发文量
105
审稿时长
4-8 weeks
期刊介绍: EMBO Molecular Medicine is an open access journal in the field of experimental medicine, dedicated to science at the interface between clinical research and basic life sciences. In addition to human data, we welcome original studies performed in cells and/or animals provided they demonstrate human disease relevance. To enhance and better specify our commitment to precision medicine, we have expanded the scope of EMM and call for contributions in the following fields: Environmental health and medicine, in particular studies in the field of environmental medicine in its functional and mechanistic aspects (exposome studies, toxicology, biomarkers, modeling, and intervention). Clinical studies and case reports - Human clinical studies providing decisive clues how to control a given disease (epidemiological, pathophysiological, therapeutic, and vaccine studies). Case reports supporting hypothesis-driven research on the disease. Biomedical technologies - Studies that present innovative materials, tools, devices, and technologies with direct translational potential and applicability (imaging technologies, drug delivery systems, tissue engineering, and AI)
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