Wassilis S. C. Bruins, Rosa Rentenaar, John Newcomb, Wenrou Zheng, Ruud W. J. Ruiter, Thomas Baardemans, Eric Poma, Chris Moore, Garrett L. Robinson, Anya Lublinsky, Yuhong Zhang, Sakeena Syed, Michael Milhollen, Ajeeta B. Dash, Niels W. C. J. van de Donk, Richard W. J. Groen, Sonja Zweegman, Tuna Mutis
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引用次数: 0
摘要
尽管多发性骨髓瘤(MM)的治疗取得了重大进展,但复发/难治患者迫切需要更有效的疗法。我们在本文中描述了工程毒素体MT-0169的发现、作用机制和临床前抗多发性骨髓瘤活性,MT-0169是一种下一代免疫毒素,由CD38特异性抗体片段与去免疫的志贺样毒素A亚基(SLTA)有效载荷连接而成。我们的研究表明,MT-0169 与 MM 细胞系上 CD38 的特异性结合会引发 SLTA 的快速内化,通过不可逆的核糖体抑制、蛋白质合成阻断和 caspase 3/7 激活导致细胞死亡。在共培养实验中,骨髓间充质基质细胞不会诱发对 MT-0169 的耐药性。在临床前研究中,MT-0169能有效裂解新诊断和重度预处理的MM患者(包括对达拉单抗难治的患者)的原发性MM细胞,对非恶性造血细胞的毒性极小。MM细胞的溶解与它们的CD38表达水平有显著相关性,但与细胞遗传风险、肿瘤负荷或之前的治疗次数无关。最后,MT-0169在各种小鼠异种移植模型中显示出高效的体内抗MM活性,其中包括MM细胞在人源化骨髓样龛中生长的模型。这些研究结果支持对复发/难治性 MM 患者(包括 CD38 靶向免疫疗法难治者)进行 MT-0169 临床研究。
Preclinical evaluation of the CD38-targeting engineered toxin body MT-0169 against multiple myeloma
Despite significant progress in the treatment of multiple myeloma (MM), relapsed/refractory patients urgently require more effective therapies. We here describe the discovery, mechanism of action, and preclinical anti-MM activity of engineered toxin body MT-0169, a next-generation immunotoxin comprising a CD38-specific antibody fragment linked to a de-immunized Shiga-like toxin A subunit (SLTA) payload. We show that specific binding of MT-0169 to CD38 on MM cell lines triggers rapid internalization of SLTA, causing cell death via irreversible ribosome inhibition, protein synthesis blockade, and caspase 3/7 activation. In co-culture experiments, bone marrow mesenchymal stromal cells did not induce drug resistance against MT-0169. In the preclinical setting, MT-0169 effectively lysed primary MM cells from newly diagnosed and heavily pretreated MM patients, including those refractory to daratumumab, with minimal toxicity against nonmalignant hematopoietic cells. MM cell lysis showed a significant correlation with their CD38 expression levels but not with cytogenetic risk, tumor load, or number of prior lines of therapy. Finally, MT-0169 showed efficient in vivo anti-MM activity in various mouse xenograft models, including one in which MM cells are grown in a humanized bone marrow-like niche. These findings support clinical investigation of MT-0169 in relapsed/refractory MM patients, including those refractory to CD38-targeting immunotherapies.
期刊介绍:
HemaSphere, as a publication, is dedicated to disseminating the outcomes of profoundly pertinent basic, translational, and clinical research endeavors within the field of hematology. The journal actively seeks robust studies that unveil novel discoveries with significant ramifications for hematology.
In addition to original research, HemaSphere features review articles and guideline articles that furnish lucid synopses and discussions of emerging developments, along with recommendations for patient care.
Positioned as the foremost resource in hematology, HemaSphere augments its offerings with specialized sections like HemaTopics and HemaPolicy. These segments engender insightful dialogues covering a spectrum of hematology-related topics, including digestible summaries of pivotal articles, updates on new therapies, deliberations on European policy matters, and other noteworthy news items within the field. Steering the course of HemaSphere are Editor in Chief Jan Cools and Deputy Editor in Chief Claire Harrison, alongside the guidance of an esteemed Editorial Board comprising international luminaries in both research and clinical realms, each representing diverse areas of hematologic expertise.