组蛋白 MARylation 通过促进 IGFBP1 甲基化调节结直肠癌的脂质代谢

IF 3.3 3区 生物学 Q3 CELL BIOLOGY
Chuanling Wang , Yi Tang , ShuXian Zhang , Ming Li , QingShu Li , Ming Xiao , Lian Yang , YaLan Wang
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引用次数: 0

摘要

在全球卫生界,结直肠癌(CRC)是一个备受关注的问题,发病率很高。单-ADP-核糖基化(MARylation)是表观遗传学的一种,被认为是导致 CRC 发展和恶化的原因之一。虽然 CRC 中的修饰水平和靶蛋白尚不清楚,但研究发现组蛋白 3 的精氨酸-117(H3R117)的 MARylation 促进了细胞的增殖,上调了肿瘤抑制基因的甲基化,并与 LoVo 细胞的代谢过程密切相关。脂质代谢紊乱参与了早期 CRC 的发病。我们的研究发现,LoVo 细胞中 H3R117 的 MARylation 调节了脂质代谢,增加了胆固醇的合成,促进了脂质筏(LR)蛋白 IGF-1R 的分布,并通过 IGFBP1 抑制了细胞凋亡。此外,生物信息学分析表明,与正常组织相比,IGFBP1 启动子在 CRC 中存在高甲基化。此外,H3R117 MARylation通过组蛋白H3瓜氨酸化(H3cit)增加了IGFBP1启动子的H3K9me2、异染色质蛋白1(HP1)和DNA甲基转移酶1(DNMT1)的富集,从而上调了IGFBP1启动子的甲基化。因此,IGFBP1 可能具有抑癌基因的功能,而 H3R117 MARylation 则可能促进 CRC 的发展。我们的研究结果丰富了现有的 CRC 表观遗传学数据,并为 H3R117 MARylation 成为 CRC 治疗靶点提供了新的思路和实验依据。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Histone MARylation regulates lipid metabolism in colorectal cancer by promoting IGFBP1 methylation
In the global health community, colorectal cancer (CRC) is a major concern, with a high rate of incidence. Mono-ADP-ribosylation (MARylation) is a type of epigenetics and recognized as one of the causes of CRC development and progression. Although the modification level and target proteins in CRC remain unclear, it has been found that MARylation of arginine-117 of histone 3 (H3R117) promotes the proliferation, upregulates methylation of tumor suppressor gene, and is tightly associated with the metabolic processes in LoVo cells. Lipid metabolism disorder is involved in the development of CRC at the early stage. Our study revealed that MARylation of H3R117 of the LoVo cells modulated lipid metabolism, increased cholesterol synthesis, promoted lipid raft (LR) protein IGF-1R distribution, and inhibited cell apoptosis through IGFBP1. In addition, bioinformatics analyses revealed that IGFBP1 promoter was hypermethylated in CRC when compared to that in normal tissues. Moreover, H3R117 MARylation upregulated the methylation of IGFBP1 promoter through histone H3 citrullination (H3cit) by increasing the H3K9me2, heterochromatin protein1 (HP1), and DNA methyltransferase 1 (DNMT1) enrichment of IGFBP1 promoter. Accordingly, IGFBP1 may function as a tumor suppressor gene, while H3R117 MARylation may promote CRC development. Our study findings enrich the available data on epigenetics of CRC and provide a new idea and experimental basis for H3R117 MARylation as a target in CRC treatment.
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来源期刊
Experimental cell research
Experimental cell research 医学-细胞生物学
CiteScore
7.20
自引率
0.00%
发文量
295
审稿时长
30 days
期刊介绍: Our scope includes but is not limited to areas such as: Chromosome biology; Chromatin and epigenetics; DNA repair; Gene regulation; Nuclear import-export; RNA processing; Non-coding RNAs; Organelle biology; The cytoskeleton; Intracellular trafficking; Cell-cell and cell-matrix interactions; Cell motility and migration; Cell proliferation; Cellular differentiation; Signal transduction; Programmed cell death.
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