二甲双胍通过 AMPK 信号减轻声带皱褶纤维化

IF 4.5 2区 医学 Q2 CELL BIOLOGY
Jie Cai, Lucheng Fang, Peng Zhou, Jianghao Wu, Yuliang Song, Aikebaier Tuohuti, Yuechen Sun, Xiong Chen
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引用次数: 0

摘要

声带纤维化是一种具有挑战性的疾病,目前尚未就有效的治疗方法达成明确共识。鉴于二甲双胍在治疗各种纤维化疾病方面的疗效,我们假设二甲双胍可通过 AMPK 信号通路减轻声带纤维化。在研究中,我们诱导兔子声带损伤,并在损伤后两周以 250 毫克/千克的剂量腹腔注射二甲双胍。损伤四周后,切除声带,使用马森氏三色染色法、免疫组化法、实时定量聚合酶链反应(qPCR)和 Western 印迹法分析声带纤维化情况。在体外,利用二甲双胍(10 μM)± TGF-β1(10 ng/mL)处理的声带成纤维细胞来评估二甲双胍的抗纤维化作用,并利用化合物 C(10 μM)抑制 AMPK 信号转导。结果表明,二甲双胍能明显改善声带薄层结构的完整性,减少胶原沉积,降低 COL1A1 和 α-SMA 的表达水平。此外,二甲双胍还能激活声带成纤维细胞中的 AMPK 信号通路,从而降低 COL1A1、α-SMA、TGF-β、Smad2 和 Smad3 的表达。这些研究结果表明,二甲双胍通过调节AMPK信号通路减轻声带纤维化,为开发治疗声带纤维化的新疗法奠定了基础。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Metformin Attenuates Vocal Fold Fibrosis via AMPK Signaling.

Vocal fold fibrosis is a challenging condition with no clear consensus on effective treatment methods. Given the demonstrated efficacy of metformin in treating various fibrotic diseases, we hypothesized that metformin could reduce vocal fold fibrosis via the AMPK signaling pathway. In our study, we induced vocal fold injury in rabbits and administered metformin intraperitoneally at a dose of 250 mg/kg two weeks post-injury. Four weeks after the injury, vocal folds were excised and analyzed for fibrosis using Masson's trichrome staining, immunohistochemistry, quantitative real-time polymerase chain reaction (qPCR), and Western blotting. In vitro, vocal fold fibroblasts treated with metformin (10 μM) ± TGF-β1 (10 ng/mL) were utilized to assess metformin's antifibrotic effects, with Compound C (10 μM) employed to inhibit AMPK signaling. Our results demonstrate that metformin significantly improved the structural integrity of the vocal fold lamina, reduced collagen deposition, and decreased the expression levels of COL1A1 and α-SMA. Furthermore, metformin activated the AMPK signaling pathway in vocal fold fibroblasts, resulting in decreased expression of COL1A1, α-SMA, TGF-β, Smad2, and Smad3. These findings suggest that metformin attenuates vocal fold fibrosis by modulating the AMPK signaling pathway, providing a foundation for developing new therapeutic options for vocal fold fibrosis.

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来源期刊
Inflammation
Inflammation 医学-免疫学
CiteScore
9.70
自引率
0.00%
发文量
168
审稿时长
3.0 months
期刊介绍: Inflammation publishes the latest international advances in experimental and clinical research on the physiology, biochemistry, cell biology, and pharmacology of inflammation. Contributions include full-length scientific reports, short definitive articles, and papers from meetings and symposia proceedings. The journal''s coverage includes acute and chronic inflammation; mediators of inflammation; mechanisms of tissue injury and cytotoxicity; pharmacology of inflammation; and clinical studies of inflammation and its modification.
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