主动脉瓣置换术后完全性和持续性房室传导阻滞的矛盾恢复:病例报告。

Pub Date : 2024-10-05 eCollection Date: 2024-10-01 DOI:10.1093/ehjcr/ytae549
Ami Nishihara, Yuta Okabe, Sei Morizumi, Yoshiharu Enomoto, Kentaro Yoshida
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引用次数: 0

摘要

背景:主动脉瓣置换术最重要和最常见的并发症之一是房室传导阻滞。病例摘要:一名 66 岁的男子在酒后行走时出现晕厥,随后来到我院就诊。病例摘要:一名 66 岁的男子在饮酒后行走时出现晕厥,随后来到我院就诊。几年前,他曾经历过两次由酒精引起的晕厥。他的心电图和经胸超声心动图分别显示完全性房室传导阻滞和严重的主动脉瓣狭窄。他在入院当天接受了临时起搏器,并在住院第 9 天接受了主动脉瓣置换手术。原主动脉瓣为双尖瓣。出乎意料的是,主动脉瓣置换术后的心电图显示,临时心房起搏时房室传导完全恢复。房室传导阻滞没有复发,他在术后第 13 天出院回家:主动脉瓣置换术后完全和持续的房室传导阻滞完全恢复,这一罕见的临床过程可由多种因素解释:(i)手术切除了直接阻碍瓣下传导系统的主动脉瓣环钙化;(ii)减轻了室间隔内传导系统所承受的过大的心室压力;以及(iii)改善了本例患者酒精敏感性晕厥所表现出的严重自主神经失调,这是减轻影响左心室机械感受器的心室内压力的结果。
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Paradoxical restoration from complete and persistent atrioventricular block after surgical aortic valve replacement: a case report.

Background: One of the most important and relatively frequent complications of aortic valve replacement is atrioventricular block. It typically occurs by direct injury of the infranodal conduction system due to intra-operative manipulation and persists post-operatively, necessitating permanent pacemaker implantation in many cases.

Case summary: A 66-year-old man presented to our hospital after experiencing syncope while walking after drinking. He had experienced two episodes of alcohol-induced syncope several years earlier. His electrocardiogram (ECG) and transthoracic echocardiogram revealed complete atrioventricular block and severe aortic stenosis, respectively. He received a temporary pacemaker on the day of admission and underwent surgical aortic valve replacement on hospital Day 9. The native aortic valve was bicuspid. Unexpectedly, the ECG immediately after aortic valve replacement showed complete restoration of atrioventricular conduction during temporary atrial pacing. The atrioventricular block did not recur, and he was discharged to home on post-operative Day 13.

Discussion: This remarkably rare clinical course, complete restoration from complete and persistent atrioventricular block after surgical aortic valve replacement, can be explained by multifactorial mechanisms: (i) surgical removal of the aortic annulus calcification directly hindering the infranodal conduction system; (ii) relief from the ventricular pressure overload stressing the conduction system within the septum; and (iii) improvement of substantial autonomic dysregulation as manifested by alcohol-sensitive syncope in the present patient, which was a result of unloading of the intraventricular pressure affecting the left ventricular mechanoreceptor.

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