新城疫病毒通过激活 VEGFR2 的 PI3K/AKT 信号通路诱导凝集素介导的内吞，从而建立感染机制

IF 4 2区医学 Q2 VIROLOGY

Journal of Virology Pub Date : 2024-09-10 DOI:10.1128/jvi.01322-24

Lei FanHongtao XiaoJinlian RenYuechi HouJuncheng CaiWanyan WuBin XiangQiuyan LinMing LiaoTao RenLibin Chen1College of Veterinary Medicine, South China Agricultural University, Guangzhou, China2Key Laboratory of Animal Vaccine Development, Ministry of Agriculture and Rural Affairs, Guangzhou, China3National and Regional Joint Engineering Laboratory for Medicament of Zoonosis Prevention and Control, Guangzhou, China4Key Laboratory of Zoonosis Prevention and Control of Guangdong Province, Guangzhou, China5College of Veterinary Medicine, Yunnan Agricultural University, Kunming, China6Zhongkai University of Agriculture and Engineering, Guangzhou, ChinaChristiane E. Wobus

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引用次数: 0

摘要

病毒学杂志》，提前出版。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Newcastle disease virus induces clathrin-mediated endocytosis to establish infection through the activation of PI3K/AKT signaling pathway by VEGFR2

Journal of Virology, Ahead of Print.

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来源期刊

Journal of Virology 医学-病毒学

CiteScore

10.10

自引率

7.40%

发文量

906

审稿时长

1 months

期刊介绍： Journal of Virology (JVI) explores the nature of the viruses of animals, archaea, bacteria, fungi, plants, and protozoa. We welcome papers on virion structure and assembly, viral genome replication and regulation of gene expression, genetic diversity and evolution, virus-cell interactions, cellular responses to infection, transformation and oncogenesis, gene delivery, viral pathogenesis and immunity, and vaccines and antiviral agents.