cathepsins 与多发性硬化症的因果关系:亡羊补牢式随机研究

Haining Lin, Yuqing Shi, Huazhong Xiong, Dongyi Wang, Shilei Wang, Hailan Kang, ZiXu Wang, Zeyu Wang, Jixiang Ren
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引用次数: 0

摘要

背景多发性硬化症(MS)是一种慢性自身免疫性神经退行性疾病。尽管有证据表明,蛋白酶可能参与了多发性硬化症的发病过程,但这种参与的确切性质仍不确定。本研究的目的是确定蛋白酶与多发性硬化症之间是否存在因果关系。方法 本研究旨在探讨九种胰蛋白酶与多发性硬化症之间的关系,并在研究设计中纳入了异质性和敏感性分析。磁共振研究按照 STROBE-MR 检查表进行报告。结果 MR 分析显示 cathepsin H 与多发性硬化症之间存在因果关系(IVW:P=0.036,OR=1.095,95% CI=1.006-1.192);chepsin E 与多发性硬化症之间存在反因果关系(IVW:P=0.031,OR=1.043,95% CI=1.004-1.083)。结论 基因预测的多发性硬化症发病风险与酪蛋白酶 H 的升高有关,而酪蛋白酶 E 的升高与多发性硬化症的发病有关,它们之间的因果关系都是单向的。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The causal relationship between cathepsins and multiple sclerosis : a mendelian randomization study
Background Multiple sclerosis (MS) is a chronic autoimmune neurodegenerative disease. Although some evidence indicates the potential involvement of cathepsins in the MS process, the precise nature of this involvement remains uncertain. The objective of this study was to ascertain whether there is a causal relationship between cathepsins and MS. Methods This study aimed to examine the relationship between nine cathepsins and MS, incorporating heterogeneity and sensitivity analyses into the study design. The MR study was reported according to the STROBE-MR checklist. Results The MR analysis revealed a causal relationship between cathepsin H and MS (IVW: P=0.036, OR=1.095, 95% CI=1.006-1.192); and an inverse causal relationship between cathepsin E and MS (IVW: P=0.031, OR=1.043, 95% CI=1.004-1.083). Conclusion Genetically predicted risk of developing MS was associated with increased cathepsin H, whereas elevated cathepsin E was associated with developing MS, and their causal relationships were both unidirectional.
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