利用高分辨率数字图像相关技术识别 AA2024-T3 疲劳裂纹生长过程中塑性诱发的裂纹闭合

Florian Paysan, David Melching, Eric Breibarth
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引用次数: 0

摘要

韧性材料的疲劳裂纹增长主要是由破坏机制和屏蔽机制之间的相互作用所驱动的。在巴黎机制中,主要的延缓机制是塑性诱导的裂纹闭合(PICC)。然而,这一现象背后的一些机制仍不清楚。在实验过程中,识别和区分三维方面与其他屏蔽方面是极其复杂的。在本文中,我们根据二维高分辨率数字图像相关数据和三维有限元模拟中的局部裂纹张开位移测量结果,对裂纹张开运动学进行了分析。结果证实,裂纹开口应力强度因子 Kop 沿裂纹路径不同。我们提出了一种确定裂纹前沿 Kop 的新方法,从而使我们能够确定 PICC 是疲劳裂纹生长实验中的主要屏蔽机制。此外,这项工作还有助于讨论 PICC 的减损效应,因为我们发现当裂纹闭合且裂纹表面接触朝向表面时,PICC 对塑性区疲劳损伤的影响仍然可以忽略不计。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Plasticity-induced crack closure identification during fatigue crack growth in AA2024-T3 by using high-resolution digital image correlation
Fatigue crack growth in ductile materials is primarily driven by the interaction between damaging and shielding mechanisms. In the Paris regime, the predominant mechanism for retardation is plasticity-induced crack closure (PICC). However, some of the mechanisms behind this phenomenon are still unclear. Identifying and separating the three-dimensional aspect from other shielding aspects during experiments is extremely complex. In this paper, we analyze the crack opening kinematics based on local crack opening displacement measurements in both 2D high-resolution digital image correlation data and 3D finite element simulations. The results confirm that the crack opening stress intensity factor Kop differs along the crack path. We present a new method to determine Kop at the crack front allowing us to identify PICC as the predominant shielding mechanism in fatigue crack growth experiments. Furthermore, this work contributes to the discussion on the damage-reducing effect of PICC, since we find that the influence on fatigue damage in the plastic zone remains negligible when the crack is closed and crack surface contact is directed towards the surface.
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