ADRA2A 可促进胰腺癌的经典/祖细胞亚型并降低疾病的侵袭性。

IF 3.3 3区 医学 Q2 ONCOLOGY
Paloma Moreno, Yuuki Ohara, Amanda J Craig, Huaitian Liu, Shouhui Yang, Tiffany H Dorsey, Lin Zhang, Gatikrushna Panigrahi, Helen Cawley, Azadeh Azizian, Jochen Gaedcke, Michael Ghadimi, Nader Hanna, S Perwez Hussain
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引用次数: 0

摘要

胰腺导管腺癌(PDAC)表现出多种分子亚型,包括经典/原发亚型和基底样/鳞状亚型,后者以其侵袭性而闻名。我们采用了转录组和代谢组的综合分析,以确定导致分子亚型分化及其代谢特征的潜在基因。我们的综合分析发现,肾上腺素受体α2A(ADRA2A)在基底样/鳞状亚型中表达下调,这表明它可能是该亚型的候选抑制因子。ADRA2A表达的降低与PDAC患者淋巴结转移频率高、病理分级高、疾病分期晚以及生存率降低有显著相关性。体外实验表明,ADRA2A 转基因表达和 ADRA2A 激动剂可抑制 PDAC 细胞侵袭。此外,在我们的 PDAC 患者群和 PDAC 细胞系中,ADRA2A 高表达条件下调了基底样/鳞状基因表达特征,同时上调了经典/祖细胞基因表达特征。对 PDAC 患者群和细胞系进行的代谢组分析表明,ADRA2A 水平的升高与氨基酸和肉碱/乙酰肉碱代谢受抑制有关,而这正是经典/祖细胞亚型的特征性代谢特征。总之,我们的研究结果表明,ADRA2A表达的升高诱导了转录组和代谢组特征,表明PDAC患者的经典/原癌基因亚型具有降低疾病侵袭性的特征。这些观察结果使 ADRA2A 成为 PDAC 诊断和治疗的候选靶点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer.

Pancreatic ductal adenocarcinoma (PDAC) manifests diverse molecular subtypes, including the classical/progenitor and basal-like/squamous subtypes, with the latter known for its aggressiveness. We employed integrative transcriptome and metabolome analyses to identify potential genes contributing to the molecular subtype differentiation and its metabolic features. Our comprehensive analysis revealed that adrenoceptor alpha 2A (ADRA2A) was downregulated in the basal-like/squamous subtype, suggesting its potential role as a candidate suppressor of this subtype. Reduced ADRA2A expression was significantly associated with a high frequency of lymph node metastasis, higher pathological grade, advanced disease stage, and decreased survival among PDAC patients. In vitro experiments demonstrated that ADRA2A transgene expression and ADRA2A agonist inhibited PDAC cell invasion. Additionally, ADRA2A-high condition downregulated the basal-like/squamous gene expression signature, while upregulating the classical/progenitor gene expression signature in our PDAC patient cohort and PDAC cell lines. Metabolome analysis conducted on the PDAC cohort and cell lines revealed that elevated ADRA2A levels were associated with suppressed amino acid and carnitine/acylcarnitine metabolism, which are characteristic metabolic profiles of the classical/progenitor subtype. Collectively, our findings suggest that heightened ADRA2A expression induces transcriptome and metabolome characteristics indicative of classical/progenitor subtype with decreased disease aggressiveness in PDAC patients. These observations introduce ADRA2A as a candidate for diagnostic and therapeutic targeting in PDAC.

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来源期刊
Carcinogenesis
Carcinogenesis 医学-肿瘤学
CiteScore
9.20
自引率
2.10%
发文量
95
审稿时长
1 months
期刊介绍: Carcinogenesis: Integrative Cancer Research is a multi-disciplinary journal that brings together all the varied aspects of research that will ultimately lead to the prevention of cancer in man. The journal publishes papers that warrant prompt publication in the areas of Biology, Genetics and Epigenetics (including the processes of promotion, progression, signal transduction, apoptosis, genomic instability, growth factors, cell and molecular biology, mutation, DNA repair, genetics, etc.), Cancer Biomarkers and Molecular Epidemiology (including genetic predisposition to cancer, and epidemiology), Inflammation, Microenvironment and Prevention (including molecular dosimetry, chemoprevention, nutrition and cancer, etc.), and Carcinogenesis (including oncogenes and tumor suppressor genes in carcinogenesis, therapy resistance of solid tumors, cancer mouse models, apoptosis and senescence, novel therapeutic targets and cancer drugs).
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