用腓骨肌腱-骨同种异体移植重建胫骨结节骨折复位犬的股四头肌伸展机制

Animals Pub Date : 2024-08-09 DOI:10.3390/ani14162315
Hyunho Kim, Haebeom Lee, Daniel D. Lewis, Jaemin Jeong, Gyumin Kim, Young-Seok Jeon
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摘要

非还原性胫骨结节骨折是在矫正犬髌骨内侧脱位(MPL)时进行胫骨结节移位术的一种罕见并发症。这种情况会严重破坏股四头肌伸展机制,导致骨盆肢体严重跛行。一只 11 岁、体重 1.8 千克的绝育雌性约克夏梗犬在接受 MPL 矫正手术时左胫骨结节粉碎性骨折。术后六个月,该犬明显跛行,左跗关节无法伸展。X 光片显示髌骨变硬,碎裂的胫骨结节吸收。手术中使用了复合冷冻异体小头腱骨块来重建胫骨结节并重新连接髌韧带。术后初步X光片显示,髌韧带与髌骨的长度比(1.42)恢复正常。用于重建胫骨结节的同种异体骨和用于重新连接髌韧带的肌腱均已成功整合。根据狗主人术后29个月的报告,这只狗恢复了令人满意的肢体功能,髌骨脱位没有复发。使用小方肌腱骨同种异体移植有效恢复了股四头肌伸展机制的功能完整性,为解决因骨性胫骨插入点缺失导致的股四头肌功能不全提供了一种可行的选择。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Reconstruction of the Quadriceps Extensor Mechanism with a Calcaneal Tendon–Bone Allograft in a Dog with a Resorbed Tibial Tuberosity Fracture
A non-reducible tibial tuberosity fracture is a rare complication of tibial tuberosity transposition performed during correcting of medial patella luxation (MPL) in dogs. This condition severely disrupts the quadriceps extensor mechanism, leading to significant pelvic limb lameness. An 11-year-old, 1.8 kg spayed female Yorkshire Terrier sustained a comminuted left tibial tuberosity fracture during surgical correction of an MPL. Six months after surgery, the dog was markedly lame and unable to extend the left stifle. Radiographs revealed patella alta and resorption of the fragmented tibial tuberosity. A composite frozen allogeneic calcaneal tendon–bone block was utilized to reconstruct the tibial tuberosity and reattach the patellar ligament. Initial postoperative radiographs confirmed restoration of a normal patellar ligament to patella length ratio (1.42). Both the allogeneic bone used for tibial tuberosity reconstruction and the tendon used to reattach the patellar ligament were successfully integrated. The dog regained satisfactory limb function without recurrence of patella luxation, as reported by the owners 29 months postoperatively. The use of a calcaneal tendon–bone allograft effectively restored the functional integrity of the quadriceps extensor mechanism, providing a viable option for addressing quadriceps insufficiency resulting from the loss of the osseous tibial insertion.
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