肾活检后肾脏诱发的突发高血压状态病例报告

Carlos Rodriguez , Dhara Rana , Ujas Shah
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引用次数: 0

摘要

背景性肾病是继发性高血压的一种罕见病因,由囊下血肿或肿块压迫肾脏导致动脉高血压升高引起。肾实质扩张性出血的常见原因是钝性外伤或较少见的先天性原因,如肾活检。大多数情况下,通过肾动脉栓塞(RAE)止血,排出血肿,继发性高血压即可缓解。病例报告我们讨论的是一名 23 岁女性,既往有高血压、慢性肾脏病病史,评估前三天曾接受过左肾活检,因左侧腹部剧烈疼痛到急诊科就诊。在急诊科进行初步评估后,影像学检查发现了一个巨大的膨胀性囊下血肿,表明是肾脏引起的高血压急症。为什么急诊医生应该注意这一点?急诊医生应该能够识别这种罕见的继发性高血压病因,以便采取适当的治疗措施。应立即进行血压控制、影像学检查和适当的会诊。与我们介绍的病例一样,确定活动性出血的来源对于明确治疗至关重要。快速识别肾脏是控制危及生命的继发性高血压、预防肾衰竭和处理任何活动性出血来源的关键。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Page kidney induced emergent hypertension status post renal biopsy a case report

Background

Page kidney is a rare cause of secondary hypertension arising from a subcapsular hematoma or mass compressing the kidney leading to increased arterial hypertension. Common causes of expansive kidney parenchymal bleeding are due to blunt trauma or less often iatrogenic causes, such as renal biopsy. Most of the time the secondary hypertension is resolved by evacuating the hematoma by stopping the bleeding via renal arterial embolization (RAE).

Case report

We discuss a 23-year-old female with a past medical history of hypertension, chronic kidney disease, status post left renal biopsy three days prior to evaluation who presented to the emergency department with severe left flank pain. From initial evaluation in the emergency department, a large expansive subcapsular hematoma was identified on imaging suggesting Page kidney causing the hypertensive emergency. The patient was managed with renal arterial embolization to control bleeding and blood pressure medications.

Why should an emergency physician be aware of this

Emergency physicians should be able to identify this rare cause of secondary hypertension to initiate proper management. Immediate blood pressure control, imaging and appropriate consultations should be initiated. Identifying sources of active bleeding is crucial for definitive management as was in the case which we presented. Quick identification of Page kidney is crucial to managing life threatening secondary hypertension, preventing renal failure, and addressing any sources of active hemorrhage.

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JEM reports
JEM reports Emergency Medicine
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