更正为沉默 PCMT1 可通过 PI3K/Akt/STMN1 途径提高乳腺癌细胞对紫杉醇的敏感性。

IF 3.2 4区医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY

Chemical Biology & Drug Design Pub Date : 2024-07-09 DOI:10.1111/cbdd.14577

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引用次数: 0

摘要

Zhang K, Li JY, Li K. Silencing PCMT1 enhanced the sensitivity of breast cancer cells to paclitaxel through the PI3K/Akt/STMN1 pathway.Chem Biol Drug Des.2024 Jun;103(6):e14559. Doi: 10.1111/cbdd.14559.PMID：38853025.第1页 "作者信息 "中的 "通讯作者：湖北十堰442000 湖北医药学院附属人民医院肿瘤科李凯"。电子邮箱：[email protected]"有误：[email protected]"有误。应改为"Correspondence Kai Li, Department of Pathology, Renmin Hospital, Hubei University of Medicine, Shiyan 442000, Hubei, China.电子邮箱：[email protected]：[email protected]。"我们对此错误深表歉意。

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Correction to Silencing PCMT1 enhances the sensitivity of breast cancer cells to paclitaxel through the PI3K/Akt/STMN1 pathway

Zhang K, Li JY, Li K. Silencing PCMT1 enhances the sensitivity of breast cancer cells to paclitaxel through the PI3K/Akt/STMN1 pathway. Chem Biol Drug Des. 2024 Jun;103(6):e14559. doi: 10.1111/cbdd.14559. PMID: 38853025.

In page 1 of Author Information section, the text “Correspondence Kai Li, Department of Oncology, Renmin Hospital, Hubei University of Medicine, Shiyan 442000, Hubei, China. Email: [email protected]” was incorrect. This should have read: “Correspondence Kai Li, Department of Pathology, Renmin Hospital, Hubei University of Medicine, Shiyan 442000, Hubei, China. Email: [email protected].”

We apologize for this error.

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来源期刊

Chemical Biology & Drug Design 医学-生化与分子生物学

CiteScore

5.10

自引率

3.30%

发文量

164

审稿时长

4.4 months

期刊介绍： Chemical Biology & Drug Design is a peer-reviewed scientific journal that is dedicated to the advancement of innovative science, technology and medicine with a focus on the multidisciplinary fields of chemical biology and drug design. It is the aim of Chemical Biology & Drug Design to capture significant research and drug discovery that highlights new concepts, insight and new findings within the scope of chemical biology and drug design.

更正为 沉默 PCMT1 可通过 PI3K/Akt/STMN1 途径提高乳腺癌细胞对紫杉醇的敏感性。

摘要

更正为沉默 PCMT1 可通过 PI3K/Akt/STMN1 途径提高乳腺癌细胞对紫杉醇的敏感性。