短式 CogDrisk 痴呆症风险评估工具的开发与并发有效性

IF 4.3 Q2 BUSINESS
Kaarin J. Anstey, M. H. Huque, S. Kootar, R. Eramudugolla, M. Li
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引用次数: 0

摘要

需要基于证据的痴呆症风险评估,以便为个人和政策层面的痴呆症风险降低干预措施提供依据。我们开发了 CogDrisk 短表(CogDrisk-SF)来评估痴呆症风险因素,以应对时间和资源有限的情况。为了评估 CogDrisk 与原始 CogDrisk 的并发有效性,我们采用重复测量、平衡设计进行了在线调查。社区居民参与者(n = 647,50.1% 为女性,平均年龄 62.2 岁,年龄范围 40-89)完成了调查。CogDrisk-SF 和 CogDrisk 的平均分(sd)分别为 9.7 (5.3) 和 9.9 (5.5)。从 CogDrisk 和 CogDrisk-SF 中获得的风险评分的类内相关性为 0.92。鱼类摄入量、失眠和抑郁的一致性分别为 79%、87% 和 89%。除孤独感(94%)、高血压(94%)、胆固醇(93%)、心房颤动(91%)和认知活动(90%)外,其他项目的一致性均为 95%。CogDrisk-SF与原始CogDrisk之间的高度一致性表明,CogDrisk-SF可用于研究和临床实践。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Development and Concurrent Validity of the Short-Form CogDrisk Dementia Risk Assessment Tool

Evidence-based dementia risk assessment is required to inform individual and policy-level dementia risk reduction interventions. We developed the CogDrisk Short Form (CogDrisk-SF) to assess dementia risk factors, for situations where time and resources are limited. To evaluate concurrent validity with the original CogDrisk, we conducted an online survey using a repeated-measures, counterbalanced design. Community dwelling participants (n = 647, 50.1% were female, mean age 62.2 years, age range 40–89) completed the survey. The mean(sd) score for CogDrisk-SF and the CogDrisk was 9.7 (5.3) and 9.9 (5.5), respectively. The intraclass correlation between the risk score obtained from CogDrisk and CogDrisk-SF was 0.92. Fish intake, insomnia and depression had percentage agreements of 79%, 87% and 89% respectively. Other items had >95% agreement except for loneliness (94%), hypertension (94%), cholesterol (93%), atrial fibrillation (91%) and cognitive activity (90%). Very high agreement between the CogDrisk-SF and original CogDrisk shows that CogDrisk-SF is valid for use in research and clinical practice.

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来源期刊
The Journal of Prevention of Alzheimer's Disease
The Journal of Prevention of Alzheimer's Disease Medicine-Psychiatry and Mental Health
CiteScore
9.20
自引率
0.00%
发文量
0
期刊介绍: The JPAD Journal of Prevention of Alzheimer’Disease will publish reviews, original research articles and short reports to improve our knowledge in the field of Alzheimer prevention including: neurosciences, biomarkers, imaging, epidemiology, public health, physical cognitive exercise, nutrition, risk and protective factors, drug development, trials design, and heath economic outcomes.JPAD will publish also the meeting abstracts from Clinical Trial on Alzheimer Disease (CTAD) and will be distributed both in paper and online version worldwide.We hope that JPAD with your contribution will play a role in the development of Alzheimer prevention.
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