在马查多-约瑟夫病突变携带者的血液样本中,全局 DNA 甲基化没有升高。

IF 2.9 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
Epigenetics Pub Date : 2024-12-01 Epub Date: 2024-06-20 DOI:10.1080/15592294.2024.2368995
Luís Teves, Ana Rosa Vieira Melo, Ana F Ferreira, Mafalda Raposo, Carolina Lemos, Conceição Bettencourt, Manuela Lima
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引用次数: 0

摘要

马查多-约瑟夫病(MJD)是一种常染色体显性脊髓小脑共济失调症(SCA),由共济失调蛋白-3(ataxin-3)中的多谷氨酰胺扩增引起,这种扩增会引发一系列致病事件,包括转录失调。MJD的基因型与表型之间的相关性并不完全,这表明MJD的发病机制还受到表观遗传修饰等其他因素的影响。众所周知,DNA 甲基化可通过基因表达调控影响神经退行性疾病的病理生理学,其他 SCA 也有甲基化增加的报道。在这项工作中,我们旨在分析 MJD 携带者的整体甲基化情况。我们对 33 名 MJD 基因突变携带者(患者和临床前受试者)和 33 名健康对照者的血液样本中的 5-mC 整体水平进行了量化,这些携带者的年龄、性别和吸烟状况与健康对照者相匹配。还对 16 名 MJD 受试者的子集进行了两个时间点的试验性随访分析。结果发现,MJD 基因突变携带者与对照组之间的 5-mC 中位数水平没有差异,也没有发现甲基化水平与临床或遗传变异之间存在相关性。此外,随着时间的推移,也没有观察到全球 5-mC 水平的变化。我们的研究结果不支持与 MJD 相关的全血甲基化水平的增加。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Global DNA methylation is not elevated in blood samples from Machado-Joseph disease mutation carriers.

Machado-Joseph disease (MJD) is an autosomal dominant spinocerebellar ataxia (SCA) caused by a polyglutamine expansion in the ataxin-3 protein, which initiates a cascade of pathogenic events, including transcriptional dysregulation. Genotype-phenotype correlations in MJD are incomplete, suggesting an influence of additional factors, such as epigenetic modifications, underlying the MJD pathogenesis. DNA methylation is known to impact the pathophysiology of neurodegenerative disorders through gene expression regulation and increased methylation has been reported for other SCAs. In this work we aimed to analyse global methylation in MJD carriers. Global 5-mC levels were quantified in blood samples of 33 MJD mutation carriers (patients and preclinical subjects) and 33 healthy controls, matched by age, sex, and smoking status. For a subset of 16 MJD subjects, a pilot follow-up analysis with two time points was also conducted. No differences were found in median global 5-mC levels between MJD mutation carriers and controls and no correlations between methylation levels and clinical or genetic variables were detected. Also, no alterations in global 5-mC levels were observed over time. Our findings do not support an increase in global blood methylation levels associated with MJD.

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来源期刊
Epigenetics
Epigenetics 生物-生化与分子生物学
CiteScore
6.80
自引率
2.70%
发文量
82
审稿时长
3-8 weeks
期刊介绍: Epigenetics publishes peer-reviewed original research and review articles that provide an unprecedented forum where epigenetic mechanisms and their role in diverse biological processes can be revealed, shared, and discussed. Epigenetics research studies heritable changes in gene expression caused by mechanisms others than the modification of the DNA sequence. Epigenetics therefore plays critical roles in a variety of biological systems, diseases, and disciplines. Topics of interest include (but are not limited to): DNA methylation Nucleosome positioning and modification Gene silencing Imprinting Nuclear reprogramming Chromatin remodeling Non-coding RNA Non-histone chromosomal elements Dosage compensation Nuclear organization Epigenetic therapy and diagnostics Nutrition and environmental epigenetics Cancer epigenetics Neuroepigenetics
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