对小鼠 7 号和 8 号染色体上的德拉韦特综合征修饰基因座进行精细绘图和候选基因分析。

IF 2.7 4区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
Mammalian Genome Pub Date : 2024-09-01 Epub Date: 2024-06-11 DOI:10.1007/s00335-024-10046-3
Nicole A Hawkins, Nathan Speakes, Jennifer A Kearney
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引用次数: 0

摘要

德拉沃特综合征是一种发育性癫痫性脑病(DEE),其特征是难治性癫痫发作、与发育、认知和运动迟缓相关的合并症,以及癫痫猝死(SUDEP)导致的高死亡率。大多数 Dravet 综合征病例都是由于 SCN1A 单倍体缺乏所致,遗传修饰因子和环境因素会影响疾病的严重程度。Scn1a杂合子缺失的小鼠模型再现了德雷维综合征的主要特征,包括癫痫发作和过早死亡;然而,严重程度因遗传背景而异。在这里,我们利用间隔特异性同源基因(ISC)作图,完善了两个德雷维生存修饰基因(Dsm)位点,即 7 号染色体上的 Dsm2 和 8 号染色体上的 Dsm3。Dsm2非常复杂,至少包括两个独立的基因座,而Dsm3则被细化为一个基因座。根据大脑表达、品系依赖性差异以及与癫痫发作或癫痫的生物学相关性,对这些细化位点内的候选修饰基因进行了优先排序。Dsm2 的高优先候选基因包括 Nav2、Ptpn5、Ldha、Dbx1、Prmt3 和 Slc6a5,而 Dsm3 只有一个高优先候选基因 Psd3。这项研究凸显了德雷维综合征复杂的遗传结构,并为潜在的调节基因提供了见解,这些基因可能会影响疾病的严重程度并成为新的治疗靶点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Fine mapping and candidate gene analysis of Dravet syndrome modifier loci on mouse chromosomes 7 and 8.

Fine mapping and candidate gene analysis of Dravet syndrome modifier loci on mouse chromosomes 7 and 8.

Dravet syndrome is a developmental and epileptic encephalopathy (DEE) characterized by intractable seizures, comorbidities related to developmental, cognitive, and motor delays, and a high mortality burden due to sudden unexpected death in epilepsy (SUDEP). Most Dravet syndrome cases are attributed to SCN1A haploinsufficiency, with genetic modifiers and environmental factors influencing disease severity. Mouse models with heterozygous deletion of Scn1a recapitulate key features of Dravet syndrome, including seizures and premature mortality; however, severity varies depending on genetic background. Here, we refined two Dravet survival modifier (Dsm) loci, Dsm2 on chromosome 7 and Dsm3 on chromosome 8, using interval-specific congenic (ISC) mapping. Dsm2 was complex and encompassed at least two separate loci, while Dsm3 was refined to a single locus. Candidate modifier genes within these refined loci were prioritized based on brain expression, strain-dependent differences, and biological relevance to seizures or epilepsy. High priority candidate genes for Dsm2 include Nav2, Ptpn5, Ldha, Dbx1, Prmt3 and Slc6a5, while Dsm3 has a single high priority candidate, Psd3. This study underscores the complex genetic architecture underlying Dravet syndrome and provides insights into potential modifier genes that could influence disease severity and serve as novel therapeutic targets.

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来源期刊
Mammalian Genome
Mammalian Genome 生物-生化与分子生物学
CiteScore
4.00
自引率
0.00%
发文量
33
审稿时长
6-12 weeks
期刊介绍: Mammalian Genome focuses on the experimental, theoretical and technical aspects of genetics, genomics, epigenetics and systems biology in mouse, human and other mammalian species, with an emphasis on the relationship between genotype and phenotype, elucidation of biological and disease pathways as well as experimental aspects of interventions, therapeutics, and precision medicine. The journal aims to publish high quality original papers that present novel findings in all areas of mammalian genetic research as well as review articles on areas of topical interest. The journal will also feature commentaries and editorials to inform readers of breakthrough discoveries as well as issues of research standards, policies and ethics.
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