DNA甲基转移酶抑制剂5-aza-4'-thio-2'-deoxycytidine可诱导C>G转换和急性淋巴白血病的发生。

IF 12.5 1区 医学 Q1 ONCOLOGY
Ryan M Bertoli, Yang Jo Chung, Michael J Difilippantonio, Anthony Wokasch, Madison R B Marasco, Haley Klimaszewski, Susannah Gammell, Yuelin J Zhu, Robert L Walker, Dengchao Cao, Ajay Khanna, Matthew J Walter, James H Doroshow, Paul S Meltzer, Peter D Aplan
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引用次数: 0

摘要

DNA 甲基转移酶抑制剂(DNMTi),最常见的是胞苷类似物,是一种能降低 5'- 胞嘧啶甲基化的化合物。基于胞嘧啶去甲基化会导致肿瘤抑制基因重新表达的假设,DNMTi 被用于临床。5-氮杂-4'-硫代-2'-脱氧胞苷(Aza TdCyd 或 ATC)是最近描述的一种硫醇取代的 DNMTi,已被证明在实体瘤模型中具有抗肿瘤活性。在这里,我们研究了 ATC 在骨髓增生异常综合征小鼠移植模型中的治疗潜力。ATC 治疗导致移植的野生型骨髓有核细胞转变为淋巴白血病,而接受 ATC 治疗的健康小鼠也患上了淋巴白血病。全外显子组测序发现了数千个获得性突变,几乎所有突变都是在特定的5'-NCG-3'背景下发生的C>G转换。这些突变涉及数十个与人类淋巴白血病有关的基因,如Notch1、Pten、Pax5、Trp53和Nf1。 用ATC进行体外处理的人类细胞也在类似的背景下出现了数千个获得性C>G反转。胞苷挽救途径的限速酶 Dck 的缺失消除了 C>G 逆转。综上所述,这些研究结果表明,ATC 具有高度渗透性的致突变和致白血病表型。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The DNA Methyltransferase Inhibitor 5-Aza-4'-thio-2'-Deoxycytidine Induces C>G Transversions and Acute Lymphoid Leukemia Development.

DNA methyltransferase inhibitors (DNMTi), most commonly cytidine analogs, are compounds that decrease 5'-cytosine methylation. DNMTi are used clinically based on the hypothesis that cytosine demethylation will lead to re-expression of tumor suppressor genes. 5-Aza-4'-thio-2'-deoxycytidine (Aza-TdCyd or ATC) is a recently described thiol-substituted DNMTi that has been shown to have anti-tumor activity in solid tumor models. In this study, we investigated the therapeutic potential of ATC in a murine transplantation model of myelodysplastic syndrome. ATC treatment led to the transformation of transplanted wild-type bone marrow nucleated cells into lymphoid leukemia, and healthy mice treated with ATC also developed lymphoid leukemia. Whole-exome sequencing revealed 1,000 acquired mutations, almost all of which were C>G transversions in a specific 5'-NCG-3' context. These mutations involved dozens of genes involved in human lymphoid leukemia, such as Notch1, Pten, Pax5, Trp53, and Nf1. Human cells treated in vitro with ATC showed 1,000 acquired C>G transversions in a similar context. Deletion of Dck, the rate-limiting enzyme for the cytidine salvage pathway, eliminated C>G transversions. Taken together, these findings demonstrate a highly penetrant mutagenic and leukemogenic phenotype associated with ATC. Significance: Treatment with a DNA methyltransferase inhibitor generates a distinct mutation signature and triggers leukemic transformation, which has important implications for the research and clinical applications of these inhibitors.

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来源期刊
Cancer research
Cancer research 医学-肿瘤学
CiteScore
16.10
自引率
0.90%
发文量
7677
审稿时长
2.5 months
期刊介绍: Cancer Research, published by the American Association for Cancer Research (AACR), is a journal that focuses on impactful original studies, reviews, and opinion pieces relevant to the broad cancer research community. Manuscripts that present conceptual or technological advances leading to insights into cancer biology are particularly sought after. The journal also places emphasis on convergence science, which involves bridging multiple distinct areas of cancer research. With primary subsections including Cancer Biology, Cancer Immunology, Cancer Metabolism and Molecular Mechanisms, Translational Cancer Biology, Cancer Landscapes, and Convergence Science, Cancer Research has a comprehensive scope. It is published twice a month and has one volume per year, with a print ISSN of 0008-5472 and an online ISSN of 1538-7445. Cancer Research is abstracted and/or indexed in various databases and platforms, including BIOSIS Previews (R) Database, MEDLINE, Current Contents/Life Sciences, Current Contents/Clinical Medicine, Science Citation Index, Scopus, and Web of Science.
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