微生物诱导的膀胱肿瘤炎症反应促进了上皮-间质转化和免疫浸润的增强。

IF 2.5 4区 生物学 Q3 CELL BIOLOGY
Physiological genomics Pub Date : 2024-08-01 Epub Date: 2024-05-29 DOI:10.1152/physiolgenomics.00032.2024
Qiang Li, Yichao Sun, Kun Zhai, Bingzhi Geng, Zhenkun Dong, Lei Ji, Hui Chen, Yan Cui
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引用次数: 0

摘要

瘤内微生物群可以调节肿瘤免疫微环境(TIME),然而,瘤内微生物群影响膀胱尿路上皮癌(UCB)TIME的潜在机制仍不清楚。为了解决这个问题,我们从癌症基因组图谱(TCGA)中收集了 402 例 UCB 患者的宿主转录组和肿瘤微生物组配对样本。我们发现,瘤内微生物组图谱与上皮-间质转化(EMT)相关基因的表达模式显著相关。此外,我们还发现肿瘤内的Lachnoclostridium属和Sutterellain属可通过诱导炎症反应间接促进EMT程序。此外,这两种瘤内细菌诱导的炎症反应进一步增强了瘤内免疫浸润,影响了患者的生存和对免疫疗法的反应。此外,348 名膀胱癌患者组成的独立免疫疗法队列也验证了我们的研究结果。总之,我们的研究阐明了瘤内微生物群影响膀胱癌TIME的潜在机制,为膀胱癌的靶向治疗提供了新的指导策略。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Microbiota-induced inflammatory responses in bladder tumors promote epithelial-mesenchymal transition and enhanced immune infiltration.

The intratumoral microbiota can modulate the tumor immune microenvironment (TIME); however, the underlying mechanism by which intratumoral microbiota influences the TIME in urothelial carcinoma of the bladder (UCB) remains unclear. To address this, we collected samples from 402 patients with UCB, including paired host transcriptome and tumor microbiome data, from The Cancer Genome Atlas (TCGA). We found that the intratumoral microbiome profiles were significantly correlated with the expression pattern of epithelial-mesenchymal transition (EMT)-related genes. Furthermore, we detected that the genera Lachnoclostridium and Sutterella in tumors could indirectly promote the EMT program by inducing an inflammatory response. Moreover, the inflammatory response induced by these two intratumoral bacteria further enhanced intratumoral immune infiltration, affecting patient survival and response to immunotherapy. In addition, an independent immunotherapy cohort of 348 patients with bladder cancer was used to validate our results. Collectively, our study elucidates the potential mechanism by which the intratumoral microbiota influences the TIME of UCB and provides a new guiding strategy for the targeted therapy of UCB.NEW & NOTEWORTHY The intratumoral microbiota may mediate the bladder tumor inflammatory response, thereby promoting the epithelial-mesenchymal transition program and influencing tumor immune infiltration.

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来源期刊
Physiological genomics
Physiological genomics 生物-生理学
CiteScore
6.10
自引率
0.00%
发文量
46
审稿时长
4-8 weeks
期刊介绍: The Physiological Genomics publishes original papers, reviews and rapid reports in a wide area of research focused on uncovering the links between genes and physiology at all levels of biological organization. Articles on topics ranging from single genes to the whole genome and their links to the physiology of humans, any model organism, organ, tissue or cell are welcome. Areas of interest include complex polygenic traits preferably of importance to human health and gene-function relationships of disease processes. Specifically, the Journal has dedicated Sections focused on genome-wide association studies (GWAS) to function, cardiovascular, renal, metabolic and neurological systems, exercise physiology, pharmacogenomics, clinical, translational and genomics for precision medicine, comparative and statistical genomics and databases. For further details on research themes covered within these Sections, please refer to the descriptions given under each Section.
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