GR/miR-421-3p/mTOR通路激活的自噬作为一种补偿机制参与了雄性胎鼠产前咖啡因暴露诱发的软骨发育不良。

Hui Han, Huasong Shi, Lingxiao Jiang, Dingmei Zhang, Hui Wang, Jing Li, Liaobin Chen
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Autophagy activated by GR/miR-421-3p/mTOR pathway as a compensatory mechanism participates in chondrodysplasia induced by prenatal caffeine exposure in male fetal rats.
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