CRPD 运动障碍治疗学前沿:从证据到治疗和应用

IF 1.9 Q3 CLINICAL NEUROLOGY
Susan L. Perlman
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引用次数: 0

摘要

遗传性共济失调没有治疗方法,也没有经过验证的延缓病情恶化的方法(没有疾病改变疗法)。后天性共济失调可能有针对潜在病因的治疗方法,可以延缓或阻止病情发展,但无法逆转已经造成的损害。特发性共济失调(遗传或获得性病因不明)也没有经证实的改变病情疗法。然而,对于任何原因导致的共济失调患者,我们总能做些什么来改善他们的生活质量--治疗相关症状、提供信息和资源、为患者和家属提供咨询、帮助解决保险和残疾问题、倾听并回答他们提出的许多问题。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
CRPD frontiers in movement disorders Therapeutics: From evidence to treatment and applications

The genetic ataxias have no cures and no proven ways to delay progression (no disease-modifying therapies). The acquired ataxias may have treatments that address the underlying cause and may slow or stop progression, but will not reverse damage already sustained. The idiopathic ataxias (of unknown genetic or acquired cause) also have no proven disease-modifying therapies. However, for all patients with ataxia of any cause, there is always something that can be done to improve quality of life—treat associated symptoms, provide information and resources, counsel patient and family, help with insurance and disability concerns, be available to listen and answer the many questions they will have.

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来源期刊
Clinical Parkinsonism  Related Disorders
Clinical Parkinsonism Related Disorders Medicine-Neurology (clinical)
CiteScore
2.70
自引率
0.00%
发文量
50
审稿时长
98 days
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