回顾:索斯比眼底营养不良症中 TIMP-3 的积累和发病机制。

IF 1.8 3区 医学 Q4 BIOCHEMISTRY & MOLECULAR BIOLOGY
Molecular Vision Pub Date : 2024-03-03 eCollection Date: 2024-01-01
Jacob H J Betts, Linda Troeberg
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引用次数: 0

摘要

索斯比眼底营养不良症(SFD)是一种罕见的遗传性黄斑变性,由编码金属蛋白酶组织抑制因子 3(TIMP-3)的基因突变引起。目前与 SFD 相关的基因突变有 21 种,对某些变异(如 Ser179Cys、Tyr191Cys 和 Ser204Cys)的研究远多于对其他变异的研究。我们从二聚化、金属蛋白酶抑制和对血管生成的影响等方面回顾了目前已知的已确定的 SFD 变体,重点是报告之间的差异和需要进一步研究的领域。我们还探讨了导致 SFD 中细胞外 TIMP-3 积累的潜在分子机制,并考虑了积累的 TIMP-3 如何导致黄斑损伤。最近有报道称,少数 SFD 患者出现了眼外病变。我们讨论了这些有趣的发现,并探讨了 TIMP-3 的广泛表达与 SFD 主要视网膜表现之间的明显差异。我们还介绍了新型实验方法(如代谢组学和干细胞模型)在研究SFD病理方面的潜在益处。因此,这篇综述强调了我们目前对SFD分子认识的差距,并提出了支持开发新型疗法的方法。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Review: Mechanisms of TIMP-3 accumulation and pathogenesis in Sorsby fundus dystrophy.

Sorsby fundus dystrophy (SFD) is a rare, inherited form of macular degeneration caused by mutations in the gene encoding tissue inhibitor of metalloproteinases 3 (TIMP-3). There are 21 mutations currently associated with SFD, with some variants (e.g., Ser179Cys, Tyr191Cys, and Ser204Cys) having been studied much more than others. We review what is currently known about the identified SFD variants in terms of their dimerization, metalloproteinase inhibition, and impact on angiogenesis, with a focus on disparities between reports and areas requiring further study. We also explore the potential molecular mechanisms leading to the accumulation of extracellular TIMP-3 in SFD and consider how accumulated TIMP-3 causes macular damage. Recent reports have identified extraocular pathologies in a small number of SFD patients. We discuss these intriguing findings and consider the apparent discrepancy between the widespread expression of TIMP-3 and the primarily retinal manifestations of SFD. The potential benefits of novel experimental approaches (e.g., metabolomics and stem cell models) in terms of investigating SFD pathology are presented. The review thus highlights gaps in our current molecular understanding of SFD and suggests ways to support the development of novel therapies.

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来源期刊
Molecular Vision
Molecular Vision 生物-生化与分子生物学
CiteScore
4.40
自引率
0.00%
发文量
25
审稿时长
1 months
期刊介绍: Molecular Vision is a peer-reviewed journal dedicated to the dissemination of research results in molecular biology, cell biology, and the genetics of the visual system (ocular and cortical). Molecular Vision publishes articles presenting original research that has not previously been published and comprehensive articles reviewing the current status of a particular field or topic. Submissions to Molecular Vision are subjected to rigorous peer review. Molecular Vision does NOT publish preprints. For authors, Molecular Vision provides a rapid means of communicating important results. Access to Molecular Vision is free and unrestricted, allowing the widest possible audience for your article. Digital publishing allows you to use color images freely (and without fees). Additionally, you may publish animations, sounds, or other supplementary information that clarifies or supports your article. Each of the authors of an article may also list an electronic mail address (which will be updated upon request) to give interested readers easy access to authors.
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