肥胖引发的 2 型糖尿病肠道低度炎症中嘌呤能信号成分的基因表达变化

IF 3 4区 医学 Q2 NEUROSCIENCES
José R. Cruz-Muñoz, Eduardo E. Valdez-Morales, Alma Barajas-Espinosa, Tonatiuh Barrios-García, Andrómeda Liñán-Rico, Raquel Guerrero-Alba
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引用次数: 0

摘要

研究发现,高脂饮食诱发的肠道低度炎症会引发慢性全身性炎症,这是肥胖症的特征之一,并且会先于胰岛素抵抗的出现,而胰岛素抵抗是引发 2 型糖尿病(T2D)的关键因素。嘌呤能信号通路的异常与炎症性肠病和其他胃肠道疾病的发病机制有关。然而,它们在与肥胖和 T2D 相关的肠道炎症中的作用仍有待探索。C57BL/6 J小鼠以食堂饮食喂养21周,并在第六周接受一次链脲佐菌素注射。通过 RT-qPCR 评估了结肠组织中嘌呤能信号成分的基因表达谱。与对照组相比,治疗组小鼠的结肠长度、粘膜和肌层厚度显著减少,同时NF-κB和IL-1β mRNA表达增加。此外,结肠 P2X2、P2X7 和 A3R 基因表达水平降低,而 P2Y2、NT5E 和 ADA 表达水平升高。总之,这些数据表明,这些嘌呤能信号成分可能在与肥胖和 T2D 相关的肠道低度炎症中发挥作用,因此可能成为治疗与这些疾病相关的代谢并发症的新型治疗靶点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Gene expression alterations of purinergic signaling components in obesity-associated intestinal low-grade inflammation in type 2 diabetes

Gene expression alterations of purinergic signaling components in obesity-associated intestinal low-grade inflammation in type 2 diabetes

Intestinal low-grade inflammation induced by a high-fat diet has been found to detonate chronic systemic inflammation, which is a hallmark of obesity, and precede the apparition of insulin resistance, a key factor for developing type 2 diabetes (T2D). Aberrant purinergic signaling pathways have been implicated in the pathogenesis of inflammatory bowel disease and other gastrointestinal diseases. However, their role in the gut inflammation associated with obesity and T2D remains unexplored. C57BL/6 J mice were fed a cafeteria diet for 21 weeks and received one injection of streptozotocin in their sixth week into the diet. The gene expression profile of purinergic signaling components in colon tissue was assessed by RT-qPCR. Compared to control mice, the treated group had a significant reduction in colonic length and mucosal and muscular layer thickness accompanied by increased NF-κB and IL-1β mRNA expression. Furthermore, colonic P2X2, P2X7, and A3R gene expression levels were lower, while the P2Y2, NT5E, and ADA expression levels increased. In conclusion, these data suggest that these purinergic signaling components possibly play a role in intestinal low-grade inflammation associated with obesity and T2D and thus could represent a novel therapeutic target for the treatment of the metabolic complications related to these diseases.

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来源期刊
Purinergic Signalling
Purinergic Signalling 医学-神经科学
CiteScore
6.60
自引率
17.10%
发文量
75
审稿时长
6-12 weeks
期刊介绍: Nucleotides and nucleosides are primitive biological molecules that were utilized early in evolution both as intracellular energy sources and as extracellular signalling molecules. ATP was first identified as a neurotransmitter and later as a co-transmitter with all the established neurotransmitters in both peripheral and central nervous systems. Four subtypes of P1 (adenosine) receptors, 7 subtypes of P2X ion channel receptors and 8 subtypes of P2Y G protein-coupled receptors have currently been identified. Since P2 receptors were first cloned in the early 1990’s, there is clear evidence for the widespread distribution of both P1 and P2 receptor subtypes in neuronal and non-neuronal cells, including glial, immune, bone, muscle, endothelial, epithelial and endocrine cells.
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