基因治疗前后受视网膜变性疾病影响的中央视觉通路。

IF 10.6 1区 医学 Q1 CLINICAL NEUROLOGY
Brain Pub Date : 2024-09-03 DOI:10.1093/brain/awae096
Manzar Ashtari, Jean Bennett, David A Leopold
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引用次数: 0

摘要

影响视网膜的遗传性疾病可导致部分或完全丧失视觉功能。勒伯氏先天性无视力症(LCA)是一种罕见的致盲疾病,通常为常染色体隐性遗传,无法治愈。研究表明,视网膜基因疗法可改善因 RPE65 基因(LCA2)突变而导致的 LCA 患者的视力。然而,人们对中枢视觉通路的活动如何受到该疾病或后续基因疗法的影响知之甚少。功能磁共振成像用于评估视网膜干预前和干预一年后皮层和皮层下视觉结构中的视网膜信号传输。fMRI范式包括15秒的闪烁(8赫兹)黑白棋盘块和15秒的空白(黑色)屏幕交错。使用一般线性模型评估大脑的视觉激活情况,并使用假发现率法校正多重比较。与对照组相比,LCA2 患者通过未经治疗的眼睛接受视觉刺激时,上丘(SC)的 fMRI 反应增强,而外侧膝状核(LGN)的活动减弱,这表明患者的视觉处理转向了视网膜直视通路(RT)。接受基因治疗后,对接受治疗的眼睛的刺激会在LGN和初级视觉皮层中引起明显更强的fMRI反应,这表明遗传视觉通路(GS)重新参与了一些活动。在所有患者中,治疗后 LGN 的 fMRI 反应与光敏感度临床测试的表现有显著相关性。我们的研究结果表明,LCA2 患者的低视力涉及视觉处理向视网膜直视通路的转移,而基因疗法部分恢复了通过 GS 通路的视觉传输。这种通过 GS 通路有选择性地增强视网膜输出的现象及其与视网膜退行性病变数年后视觉表现改善的相关性令人震惊。然而,虽然视网膜基因疗法和其他眼部干预措施给 RPE65 患者带来了希望,但要开发出专门用于治疗其他低视力患者疾病的疗法可能还需要数年时间。我们的研究表明,这些患者的视力会向 RT 途径转变,这可能会促进新工具和康复策略的开发,帮助他们最大限度地利用残余视觉能力,增强依赖经验的可塑性。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Central visual pathways affected by degenerative retinal disease before and after gene therapy.

Genetic diseases affecting the retina can result in partial or complete loss of visual function. Leber's congenital amaurosis (LCA) is a rare blinding disease, usually inherited in an autosomally recessive manner, with no cure. Retinal gene therapy has been shown to improve vision in LCA patients caused by mutations in the RPE65 gene (LCA2). However, little is known about how activity in central visual pathways is affected by the disease or by subsequent gene therapy. Functional MRI (fMRI) was used to assess retinal signal transmission in cortical and subcortical visual structures before and 1 year after retinal intervention. The fMRI paradigm consisted of 15-s blocks of flickering (8 Hz) black and white checkerboards interleaved with 15 s of blank (black) screen. Visual activation in the brain was assessed using the general linear model, with multiple comparisons corrected using the false discovery rate method. Response to visual stimulation through untreated eyes of LCA2 patients showed heightened fMRI responses in the superior colliculus and diminished activities in the lateral geniculate nucleus (LGN) compared to controls, indicating a shift in the patients' visual processing towards the retinotectal pathway. Following gene therapy, stimuli presented to the treated eye elicited significantly stronger fMRI responses in the LGN and primary visual cortex, indicating some re-engagement of the geniculostriate pathway (GS) pathway. Across patients, the post-treatment LGN fMRI responses correlated significantly with performance on a clinical test measuring light sensitivity. Our results demonstrate that the low vision observed in LCA2 patients involves a shift in visual processing toward the retinotectal pathway, and that gene therapy partially reinstates visual transmission through the GS pathway. This selective boosting of retinal output through the GS pathway and its correlation to improved visual performance, following several years of degenerative retinal disease, is striking. However, while retinal gene therapy and other ocular interventions have given hope to RPE65 patients, it may take years before development of therapies tailored to treat the diseases in other low vision patients are available. Our demonstration of a shift toward the retinotectal pathway in these patients may spur the development of new tools and rehabilitation strategies to help maximize the use of residual visual abilities and augment experience-dependent plasticity.

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来源期刊
Brain
Brain 医学-临床神经学
CiteScore
20.30
自引率
4.10%
发文量
458
审稿时长
3-6 weeks
期刊介绍: Brain, a journal focused on clinical neurology and translational neuroscience, has been publishing landmark papers since 1878. The journal aims to expand its scope by including studies that shed light on disease mechanisms and conducting innovative clinical trials for brain disorders. With a wide range of topics covered, the Editorial Board represents the international readership and diverse coverage of the journal. Accepted articles are promptly posted online, typically within a few weeks of acceptance. As of 2022, Brain holds an impressive impact factor of 14.5, according to the Journal Citation Reports.
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